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miRBase |
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![]() 11 publications mentioning hsa-mir-545Open access articles that are associated with the species Homo sapiens and mention the gene name mir-545. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary. |
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Other miRNAs from this paper: hsa-mir-17, hsa-mir-92a-1, hsa-mir-92a-2, hsa-mir-208a, hsa-mir-1-2, hsa-mir-133a-1, hsa-mir-133a-2, hsa-mir-145, hsa-mir-126, hsa-mir-1-1, hsa-mir-155, hsa-mir-340, hsa-mir-451a, hsa-mir-499a, hsa-mir-615, hsa-mir-624, hsa-mir-454, hsa-mir-663b, hsa-mir-1291, hsa-mir-451b, hsa-mir-499b
After all the analysis, six out of 9 miRNAs remained significantly and more than 1.5 fold upregulated (miR340*, miR615-5p, miR545:9.1, miR451, miR454* and miR624*) and 1 out of 5 miRNA remained significantly and more than 1.5 fold downregulated (miR-1280) in both analyses (figure 2).
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Six platelet microRNAs were significantly upregulated (miR340*, miR451, miR454*, miR545:9.1. miR615-5p and miR624*) and one miRNA (miR1280) was significantly downregulated in patients with CAD as compared to healthy controls.
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Other miRNAs from this paper: hsa-let-7c, hsa-mir-29a, hsa-mir-221, hsa-mir-126, hsa-mir-146a, hsa-mir-363, hsa-mir-151a, hsa-mir-429, hsa-mir-483, hsa-mir-485, hsa-mir-486-1, hsa-mir-511, hsa-mir-146b, hsa-mir-517b, hsa-mir-500a, hsa-mir-455, hsa-mir-573, hsa-mir-656, hsa-mir-668, hsa-mir-151b, hsa-mir-770, hsa-mir-888, hsa-mir-541, hsa-mir-876, hsa-mir-374b, hsa-mir-216b, hsa-mir-1285-1, hsa-mir-1285-2, hsa-mir-1284, hsa-mir-1255b-1, hsa-mir-1255b-2, hsa-mir-500b, hsa-mir-486-2
E3 ligases and deubiquitinases such as MDM2, SMUR1, NEDD4L, NEDD4, RNF41, ITCH, CBL, UBPY, and AMSH were targeted by 9 (hsa-miR-545, hsa-miR-541, hsa-miR-511, hsa-miR-888, hsa-miR-146-5p, hsamiR- 429, hsa-miR-374b, hsa-miR-500, and hsa-miR-656) of the predicted poor prognosis miRs.
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Notably, 3 of these miRs (hsa-miR-545, hsa-miR-541, and hsa-miR-511) targeted more than one component of the UPS system.
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There were 8 common miRs (hsa-miR-429, hsa-miR-221, hsa-miR- 499-5p, hsa-miR-888, hsa-miR-770-5p, hsa-miR-545, hsa-miR-541, hsa-miR-483-3p) between the two sets of survival -associated miRs (Figure S2).
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Strikingly, 9 of the poor prognosis associated miRs: hsa-miR-545, hsa-miR-541, hsa-miR-511, hsa-miR-888, hsa-miR-146- 5p, hsa-miR-429, hsa-miR-374b, hsa-miR-500, and hsamiR- 656 have functions in E3 ligases and deubiquitinases (Figure 4B).
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Gender differences in miR-545/374 a expression are recognized with male HCC patients having higher expression than females [15].
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The Ftx transcript encodes miR-545/374 a, both of which showed increased expression in tumor tissue compared with noncancerous tissue taken from patients with HBV-related HCC [15].
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Other miRNAs from this paper: hsa-mir-15a, hsa-mir-16-1, hsa-mir-16-2, mmu-mir-146a, mmu-mir-186, hsa-mir-146a, hsa-mir-186, mmu-mir-15a, mmu-mir-16-1, mmu-mir-16-2, hsa-mir-146b, mmu-mir-146b
Previous studies have shown that CCND1 is the direct target of miR-186 [33] and miR-545 [34], while both CCND1 and CCND2 are directly targeted by miR-15a and miR-16 [35].
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Other miRNAs from this paper: hsa-mir-101-1, hsa-mir-137, hsa-mir-145, hsa-mir-101-2, hsa-mir-335, hsa-mir-384, hsa-mir-451a, hsa-mir-558, hsa-mir-451b
We found several miRNAs (miR-101, miR-137, miR-145, miR-335, miR-384, miR-451, miR-545 and miR-558) upregulated in virus-transduced cells that showed resistance to 5-Aza-dC -induced senescence (Fig. 1 and Supplementary Fig. 1A,B).
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Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-15a, hsa-mir-16-1, hsa-mir-17, hsa-mir-21, hsa-mir-24-1, hsa-mir-24-2, hsa-mir-25, hsa-mir-26a-1, hsa-mir-26b, hsa-mir-29a, hsa-mir-30a, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-107, hsa-mir-192, hsa-mir-148a, hsa-mir-30c-2, hsa-mir-30d, hsa-mir-10b, hsa-mir-34a, hsa-mir-203a, hsa-mir-204, hsa-mir-210, hsa-mir-212, hsa-mir-215, hsa-mir-221, hsa-mir-223, hsa-mir-200b, hsa-let-7g, hsa-let-7i, hsa-mir-15b, hsa-mir-30b, hsa-mir-122, hsa-mir-143, hsa-mir-145, hsa-mir-9-1, hsa-mir-9-2, hsa-mir-9-3, hsa-mir-127, hsa-mir-194-1, hsa-mir-200c, hsa-mir-155, hsa-mir-194-2, hsa-mir-29c, hsa-mir-30c-1, hsa-mir-200a, hsa-mir-302a, hsa-mir-34b, hsa-mir-34c, hsa-mir-30e, hsa-mir-26a-2, hsa-mir-363, hsa-mir-365a, hsa-mir-365b, hsa-mir-302b, hsa-mir-302c, hsa-mir-302d, hsa-mir-135b, hsa-mir-424, hsa-mir-20b, hsa-mir-486-1, hsa-mir-491, hsa-mir-519c, hsa-mir-590, hsa-mir-605, hsa-mir-663a, hsa-mir-663b, hsa-mir-1246, hsa-mir-302e, hsa-mir-302f, hsa-mir-203b, hsa-mir-486-2
For examples, miRNA-545 leads to cell-cycle arrest in lung cancer cells by repressing expression of cyclin D1 and CDK4.
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Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-mir-15a, hsa-mir-18a, hsa-mir-33a, hsa-mir-103a-2, hsa-mir-103a-1, hsa-mir-107, mmu-mir-27b, mmu-mir-126a, mmu-mir-128-1, mmu-mir-140, mmu-mir-146a, mmu-mir-152, mmu-mir-155, mmu-mir-191, hsa-mir-10a, hsa-mir-211, hsa-mir-218-1, hsa-mir-218-2, mmu-mir-297a-1, mmu-mir-297a-2, hsa-mir-27b, hsa-mir-128-1, hsa-mir-140, hsa-mir-152, hsa-mir-191, hsa-mir-126, hsa-mir-146a, mmu-let-7a-1, mmu-let-7a-2, mmu-mir-15a, mmu-mir-18a, mmu-mir-103-1, mmu-mir-103-2, mmu-mir-342, hsa-mir-155, mmu-mir-107, mmu-mir-10a, mmu-mir-218-1, mmu-mir-218-2, mmu-mir-33, mmu-mir-211, hsa-mir-374a, hsa-mir-342, gga-mir-33-1, gga-let-7a-3, gga-mir-155, gga-mir-18a, gga-mir-15a, gga-mir-218-1, gga-mir-103-2, gga-mir-107, gga-mir-128-1, gga-mir-140, gga-let-7a-1, gga-mir-146a, gga-mir-103-1, gga-mir-218-2, gga-mir-126, gga-let-7a-2, gga-mir-27b, mmu-mir-466a, mmu-mir-467a-1, hsa-mir-499a, hsa-mir-593, hsa-mir-600, hsa-mir-33b, gga-mir-499, gga-mir-211, gga-mir-466, mmu-mir-675, mmu-mir-677, mmu-mir-467b, mmu-mir-297b, mmu-mir-499, mmu-mir-717, hsa-mir-675, mmu-mir-297a-3, mmu-mir-297a-4, mmu-mir-297c, mmu-mir-466b-1, mmu-mir-466b-2, mmu-mir-466b-3, mmu-mir-466c-1, mmu-mir-466e, mmu-mir-466f-1, mmu-mir-466f-2, mmu-mir-466f-3, mmu-mir-466g, mmu-mir-466h, mmu-mir-467c, mmu-mir-467d, mmu-mir-466d, hsa-mir-297, mmu-mir-467e, mmu-mir-466l, mmu-mir-466i, mmu-mir-466f-4, mmu-mir-466k, mmu-mir-467f, mmu-mir-466j, mmu-mir-467g, mmu-mir-467h, hsa-mir-664a, hsa-mir-1306, hsa-mir-1307, gga-mir-1306, hsa-mir-103b-1, hsa-mir-103b-2, gga-mir-10a, mmu-mir-1306, mmu-mir-3064, mmu-mir-466m, mmu-mir-466o, mmu-mir-467a-2, mmu-mir-467a-3, mmu-mir-466c-2, mmu-mir-467a-4, mmu-mir-466b-4, mmu-mir-467a-5, mmu-mir-466b-5, mmu-mir-467a-6, mmu-mir-466b-6, mmu-mir-467a-7, mmu-mir-466b-7, mmu-mir-467a-8, mmu-mir-467a-9, mmu-mir-467a-10, mmu-mir-466p, mmu-mir-466n, mmu-mir-466b-8, hsa-mir-466, hsa-mir-3173, hsa-mir-3618, hsa-mir-3064, hsa-mir-499b, mmu-mir-466q, hsa-mir-664b, gga-mir-3064, mmu-mir-126b, gga-mir-33-2, mmu-mir-3618, mmu-mir-466c-3, gga-mir-191
Long ncRNAs were found to also host clusters of miRNA genes and therefore encode polycistronic primary transcripts that can yield several miRNAs; for example lincRNA FTX (FTX transcript, XIST regulator (non-protein coding)) comprises two miRNA genes: hsa-mir-374a and hsa-mir-545 (Figure 3B ).
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Some miRNA genes were found to form clusters within hosting ncRNA genes: for example the miRNA gene cluster, comprising hsa-mir-374a and hsa-mir-545, is located within lincRNA gene FTX (Figure 3B ).
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Other miRNAs from this paper: hsa-let-7b, hsa-mir-32, hsa-mir-147a, hsa-mir-483, hsa-mir-493, hsa-mir-520h, hsa-mir-539, hsa-mir-574, hsa-mir-660, hsa-mir-877, hsa-mir-1228, hsa-mir-1234, hsa-mir-1238, hsa-mir-1277, hsa-mir-1281, hsa-mir-4271, hsa-mir-4290, hsa-mir-3620, hsa-mir-4644, hsa-mir-4649, hsa-mir-4716, hsa-mir-6124
miRNA Sequence miR-574-5pUGA GUGUGUGUGUGUGA GUGUGU miR-941CACCCGGCU GUGUGCACAU GUGC miR-3149UUU GUAUGGAUAU GUGUGUGUAU miR-1238-5p GUGA GUGGGAGCCCCA GUGUGUG miR-545-3pUCAGCAAACAUUUAU UGUGUGC miR-2278GAGAGCA GUGUGUGUUGCCUGG miR-3148UGGAAAAAACUG GUGUGUGCUU let-7b-5pUGAG GUA GUAG GUU GUGUG GUU miR-493-3pUGAAG GUCUACU GUGUGCCAGG miR-1180UUUCCGGCUCGC GUGG GUGUGU miR-539-5pGGAGAAAUUAUCCUUG GUGUGU miR-32-3pCAAUUUA GUGUGUGUGAUAUUU miR-206UGGAAU GUAAGGAA GUGUGUGG miR-1299UUCUGGAAUUC UGUGUGAGGGA miR-3911U GUGUGGAUCCUGGAGGAGGCA miR-297AUGUAU GUGUGCAU GUGCAUG miR-610UGAGCUAAAU GUGUGCUGGGA miR-1228-5p GUGGGCGGGGGCAG GUGUGUG miR-595GAA GUGUGCC GUG GUGUGUCU miR-4455AGG GUGUGUGUGUUUUU miR-3650AG GUGUGUCU GUAGA GUCC miR-147a GUGUGUGGAAAUGCUUCUGC miR-660-3pACCUCCU GUGUGCAUGGAUUA Interestingly, most of the trinucleotide repeats contain base “U” and “G”, although it can be noticed that this type of SSR is less represented.
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miRNA Sequence miR-574-5pUGA GUGUGUGUGUGUGA GUGUGU miR-941CACCCGGCU GUGUGCACAU GUGC miR-3149UUU GUAUGGAUAU GUGUGUGUAU miR-1238-5p GUGA GUGGGAGCCCCA GUGUGUG miR-545-3pUCAGCAAACAUUUAU UGUGUGC miR-2278GAGAGCA GUGUGUGUUGCCUGG miR-3148UGGAAAAAACUG GUGUGUGCUU let-7b-5pUGAG GUA GUAG GUU GUGUG GUU miR-493-3pUGAAG GUCUACU GUGUGCCAGG miR-1180UUUCCGGCUCGC GUGG GUGUGU miR-539-5pGGAGAAAUUAUCCUUG GUGUGU miR-32-3pCAAUUUA GUGUGUGUGAUAUUU miR-206UGGAAU GUAAGGAA GUGUGUGG miR-1299UUCUGGAAUUC UGUGUGAGGGA miR-3911U GUGUGGAUCCUGGAGGAGGCA miR-297AUGUAU GUGUGCAU GUGCAUG miR-610UGAGCUAAAU GUGUGCUGGGA miR-1228-5p GUGGGCGGGGGCAG GUGUGUG miR-595GAA GUGUGCC GUG GUGUGUCU miR-4455AGG GUGUGUGUGUUUUU miR-3650AG GUGUGUCU GUAGA GUCC miR-147a GUGUGUGGAAAUGCUUCUGC miR-660-3pACCUCCU GUGUGCAUGGAUUAInterestingly, most of the trinucleotide repeats contain base “U” and “G”, although it can be noticed that this type of SSR is less represented.
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Other miRNAs from this paper: hsa-let-7e, hsa-mir-21, hsa-mir-23a, hsa-mir-24-1, hsa-mir-24-2, hsa-mir-30a, hsa-mir-31, hsa-mir-183, hsa-mir-203a, hsa-mir-205, hsa-mir-210, hsa-mir-221, hsa-mir-222, hsa-mir-23b, hsa-mir-125b-1, hsa-mir-130a, hsa-mir-125b-2, hsa-mir-193b, hsa-mir-660, hsa-mir-421, hsa-mir-762, hsa-mir-1202, hsa-mir-4291, hsa-mir-23c, hsa-mir-203b
In this network, ten genes (SOCS6, SMAD2, CDKN2B, PPARGC1A, FOS, FOSL2, IL8, IRS2, JAK1, WDR32) were coregulated by six miRNAs (miRNA-23a, miRNA-24, miRNA-30a, miRNA-545, miRNA-203, miRNA-660) (Figure 2, Table 4).
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Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-15a, hsa-mir-16-1, hsa-mir-17, hsa-mir-18a, hsa-mir-19b-2, hsa-mir-20a, hsa-mir-21, hsa-mir-22, hsa-mir-26a-1, hsa-mir-29a, hsa-mir-92a-1, hsa-mir-92a-2, hsa-mir-93, hsa-mir-99a, hsa-mir-101-1, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-106a, hsa-mir-16-2, hsa-mir-197, hsa-mir-199a-1, hsa-mir-148a, hsa-mir-30c-2, hsa-mir-10a, hsa-mir-34a, hsa-mir-182, hsa-mir-199a-2, hsa-mir-205, hsa-mir-210, hsa-mir-221, hsa-mir-223, hsa-let-7g, hsa-let-7i, hsa-mir-15b, hsa-mir-23b, hsa-mir-122, hsa-mir-124-1, hsa-mir-124-2, hsa-mir-124-3, hsa-mir-125b-1, hsa-mir-132, hsa-mir-133a-1, hsa-mir-133a-2, hsa-mir-140, hsa-mir-142, hsa-mir-143, hsa-mir-125b-2, hsa-mir-134, hsa-mir-146a, hsa-mir-150, hsa-mir-206, hsa-mir-155, hsa-mir-29c, hsa-mir-30c-1, hsa-mir-101-2, hsa-mir-130b, hsa-mir-26a-2, hsa-mir-361, hsa-mir-362, hsa-mir-363, hsa-mir-376c, hsa-mir-371a, hsa-mir-375, hsa-mir-376a-1, hsa-mir-378a, hsa-mir-342, hsa-mir-151a, hsa-mir-324, hsa-mir-335, hsa-mir-345, hsa-mir-423, hsa-mir-483, hsa-mir-486-1, hsa-mir-146b, hsa-mir-202, hsa-mir-432, hsa-mir-494, hsa-mir-495, hsa-mir-193b, hsa-mir-497, hsa-mir-455, hsa-mir-376a-2, hsa-mir-487b, hsa-mir-551a, hsa-mir-571, hsa-mir-574, hsa-mir-576, hsa-mir-606, hsa-mir-628, hsa-mir-629, hsa-mir-411, hsa-mir-671, hsa-mir-320b-1, hsa-mir-320c-1, hsa-mir-320b-2, hsa-mir-378d-2, hsa-mir-889, hsa-mir-876, hsa-mir-744, hsa-mir-885, hsa-mir-920, hsa-mir-937, hsa-mir-297, hsa-mir-1233-1, hsa-mir-1260a, hsa-mir-664a, hsa-mir-320c-2, hsa-mir-2861, hsa-mir-378b, hsa-mir-1260b, hsa-mir-378c, hsa-mir-1233-2, hsa-mir-378d-1, hsa-mir-378e, hsa-mir-378f, hsa-mir-378g, hsa-mir-378h, hsa-mir-378i, hsa-mir-664b, hsa-mir-378j, hsa-mir-486-2
In addition, a panel comprising miR-143, miR-324-3p, and miR-545 had moderate ability in discriminating patients infected with CVA16 from those with EV71 (215).
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Other miRNAs from this paper: hsa-mir-204, hsa-mir-133a-1, hsa-mir-133a-2, hsa-mir-135a-1, hsa-mir-135a-2, hsa-mir-140, hsa-mir-135b, hsa-mir-133b, hsa-mir-675
Moreover, BACE1-AS act as a sponge on miR-545-5p and stabilizes BACE-1 mRNA levels to raise αβ peptide production [93].
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