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4 publications mentioning csi-MIR395b

Open access articles that are associated with the species Citrus sinensis and mention the gene name MIR395b. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

1
[+] score: 64
The adaptive responses of leaf miRNAs to Mg-deficiency might include following several aspects: (a) inducing stress-related genes by repressing miR164, miR7812, miR5742, miR3946, and miR5158; (b) up -regulating transport-related genes; (c) increasing the expression of genes related to lipid metabolism by inhibiting miR158, miR5256, and miR3946 expression; (d) activating cell wall-related gene expansis 8A by down -regulating miR779; and (e) down -regulating the expression of genes involved in the maintenance of S, K and Cu by up -regulating miR395 and miR6426. [score:13]
By contrast, the expression levels of miR5821, miR395, miR946, miR1160, and miR6218 were increased in Mg -deficient leaves, and their some target genes related to transport [i. e., cation efflux family protein, K [+] uptake transporter 3 (KUT3), transporter associated with antigen processing protein 1, mitochondrial substrate carrier family protein and glutamate receptor 5 (GLR5)] were down-regulated in these leaves. [score:8]
Similarly, the expression levels of the other transport-related genes targeted by miR395 and miR1077 (SecY protein transport family protein), miR1160 (PHO1), miR8019 (PPI1), and miR3946 (BAT1) were up-regulated in Mg -deficient leaves (Table 2). [score:8]
MiR395-over -expressing Arabidopsis exhibited remarkable down-regulation in mRNA levels of its two target genes, and had more accumulation of S in the shoot but not in the root. [score:7]
Also, K [+] uptake transporter 3 (KUP3), a target gene of miR395 was inhibited in Mg -deficient leaves (Table 2). [score:5]
Therefore, Mg-deficiency -induced up-regulation of leaf miR395 might contribute to the homeostasis of S in plants, which agrees with our data that Mg-deficiency did not significantly affect S concentration in C. sinensis roots, stems and leaves (Xu, 2015). [score:4]
MiR395 might play a role in the regulation of plant S accumulation and allocation by targeting APS and SULTR2;1 (Liang et al., 2010). [score:3]
The aps1-1 sultr2;1 APS4 -RNAi mutants displayed similar phenotypes to those of miR395-over -expressing plants. [score:3]
Their transcripts were decreased in transgenic Arabidopsis over -expressing miR395 accompanied by increased accumulation of S in the shoot but not in the root. [score:3]
MiR395, which targets two sulfur (S) metabolism-related genes [i. e., ATP sulfurylases (APS) and sulfate transporter 2;1 (SULTR2;1)], was induced by S-deprivation. [score:2]
These authors concluded that miR395 -mediated regulation of APS and SULTR2;1 might play a crucial role in plant S homeostasis (Liang et al., 2010). [score:2]
MiR395 targets APS1, APS2, APS4 and sulfate transporter 2;1 (SULTR2;1). [score:2]
MicroRNA395 mediates regulation of sulfate accumulation and allocation in Arabidopsis thaliana. [score:1]
Also, many other miRNAs such as miR156, miR159, miR166, miR169, miR395, miR397, miR398, miR408, miR447, miR482, miR1510 and miR2109 are involved in plant response to P-limitation (Valdés-López et al., 2010; Hackenberg et al., 2013; Zhao et al., 2013; Paul et al., 2015). [score:1]
Our finding that miR395 was induced in Mg -deficient leaves (Table 2) agrees with the report that miR395 in Arabidopsis leaves was enhanced by S-deficiency. [score:1]
Thus, it is reasonable to assume that miR395 played a role in the maintenance of S and K homeostasis. [score:1]
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2
[+] score: 15
Their transcripts are greatly down-regulated in miR395-over -expressing transgenic Arabidopsis accompanied by increased accumulation of S in the shoot but not in the root. [score:6]
They concluded that miR395 play a role in the regulation of plant S accumulation and allocation by targeting APS and SULTR2;1 [24]. [score:4]
MiR395 targets ATP sulfurylases (APS) and sulfate transporter 2;1 (SULTR2;1), both of which are involved in the S metabolism. [score:2]
It is worth noting that some N-deficiency-responsive miRNAs (e. g., miR169, miR172, miR394, miR395, miR397, miR398, miR399, miR827, miR408 and miR857) are also responsive to other nutrient stresses (i. e., B, P, Fe, S and Cu deficiencies) in plants [8, 10], indicating the involvement of miRNA -mediated crosstalk among N, B, P, Fe, S and Cu under N-deficiency. [score:1]
In addition, many other P-deficiency-responsive miRNAs (i. e., miR1510, miR156, miR159, miR166, miR169, miR2109, miR395, miR397, miR398, miR408, miR447 and miR482) have been isolated from various plant species [15– 21]. [score:1]
In Arabidopsis, leaf miR395 was induced by sulfur (S)-deficiency. [score:1]
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3
[+] score: 8
miR395 has been shown to mediate regulation of sulfate accumulation and allocation by targeting APS and SULTR2;1, respectively [12]. [score:4]
However, miR395 targets members of the ATP sulfurylase (APS) gene family [14] and the sulfate transporter SULTR2;1 [26]. [score:3]
In A. thaliana, miR395 is enhanced during sulfate-limitation, and its induction is controlled by a key transcription factor (SLIM1) in the S assimilation pathway [26]. [score:1]
[1 to 20 of 3 sentences]
4
[+] score: 3
In Arabidopsis, miR395 targets genes belonging to the ATP sulfurylase gene family 61 as well as sulfate transporters 55 to modulates sulfate accumulation and allocation 34. [score:3]
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