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![]() 4 publications mentioning csi-MIR399cOpen access articles that are associated with the species Citrus sinensis and mention the gene name MIR399c. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary. |
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Other miRNAs from this paper: csi-MIR166e, csi-MIR398a, csi-MIR166a, csi-MIR156a, csi-MIR166c, csi-MIR394a, csi-MIR395a, csi-MIR397, csi-MIR399d, csi-MIR399a, csi-MIR399b, csi-MIR408, csi-MIR857, csi-MIR3954, csi-MIR156b, csi-MIR156c, csi-MIR156d, csi-MIR156e, csi-MIR156f, csi-MIR156g, csi-MIR166b, csi-MIR166d, csi-MIR166f, csi-MIR166g, csi-MIR166h, csi-MIR166i, csi-MIR166j, csi-MIR166k, csi-MIR394b, csi-MIR395b, csi-MIR395c, csi-MIR398b, csi-MIR399e, csi-MIR399f, csi-MIR156h, csi-MIR156i, csi-MIR156j
miR399 is up-regulated in P -deficient roots and suppressed in P-sufficient roots and is negatively correlated with that of its target gene UBC24[15, 16].
[score:8]
In addition, the expression level of miR399 in barley roots and leaves was differentially regulated by B-toxicity.
[score:4]
Phenotypes of both the Arabidopsis T-DNA knockout ucb24 mutants and the miR399 -overexpressing transgenic Arabidopsis plants resemble those of a previously reported pho2 mutant, a P overaccumulator [19].
[score:4]
The inverse relationship of expression patterns between UBC24 homologs and miR399 under P-deficiency has been confirmed in common bean [17] and rice [18].
[score:3]
Transgenic Arabidopsis overexpressing miR399 also had decreased level of UBC24 transcripts [15].
[score:3]
In A. thaliana, miR399 has been predicted to target three genes, which encode a phosphate transporter (PHT1;7), a ubiquitin-conjugating E2 enzyme, and a DEAD box helicase; however, only the E2 enzyme encoded by UBC24 has been experimentally validated [14].
[score:3]
Therefore, miR399 plays important roles in maintaining P homeostasis by regulating UBC24 transcript levels [20].
[score:2]
[1 to 20 of 7 sentences]
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Other miRNAs from this paper: csi-MIR160a, csi-MIR164a, csi-MIR166e, csi-MIR169a, csi-MIR172a, csi-MIR398a, csi-MIR166a, csi-MIR156a, csi-MIR3946, csi-MIR159a, csi-MIR166c, csi-MIR167c, csi-MIR167b, csi-MIR171b, csi-MIR171a, csi-MIR172c, csi-MIR159d, csi-MIR393a, csi-MIR394a, csi-MIR395a, csi-MIR397, csi-MIR399d, csi-MIR399a, csi-MIR399b, csi-MIR408, csi-MIR482f, csi-MIR477c, csi-MIR477a, csi-MIR477b, csi-MIR482a, csi-MIR482b, csi-MIR827, csi-MIR857, csi-MIR482c, csi-MIR3953, csi-MIR167a, csi-MIR156b, csi-MIR156c, csi-MIR156d, csi-MIR156e, csi-MIR156f, csi-MIR156g, csi-MIR159b, csi-MIR159c, csi-MIR160b, csi-MIR160c, csi-MIR164b, csi-MIR164c, csi-MIR166b, csi-MIR166d, csi-MIR166f, csi-MIR166g, csi-MIR166h, csi-MIR166i, csi-MIR166j, csi-MIR166k, csi-MIR167d, csi-MIR167e, csi-MIR169b, csi-MIR169c, csi-MIR169d, csi-MIR169e, csi-MIR169f, csi-MIR169g, csi-MIR169h, csi-MIR169i, csi-MIR169j, csi-MIR169k, csi-MIR169l, csi-MIR169m, csi-MIR169n, csi-MIR171c, csi-MIR171d, csi-MIR171e, csi-MIR171f, csi-MIR171g, csi-MIR171h, csi-MIR171i, csi-MIR172b, csi-MIR172d, csi-MIR393b, csi-MIR393c, csi-MIR394b, csi-MIR395b, csi-MIR395c, csi-MIR398b, csi-MIR399e, csi-MIR399f, csi-MIR477d, csi-MIR477e, csi-MIR482d, csi-MIR482e, csi-MIR482g, csi-MIR156h, csi-MIR156i, csi-MIR156j, csi-MIR164d, csi-MIR169o, csi-MIR169p, csi-MIR169q, csi-MIR169r
Like miR399, miR827 is also highly and specifically induced by P-deficiency and is involved in the regulation of plant P homeostasis by down -regulating its target gene nitrogen limitation adaptation (NLA) in Arabidopsis [13].
[score:5]
MiR399, which is specifically induced by P-deficiency in Arabidopsis and rice, can regulate P homeostasis by negatively regulating its target gene UBC24 [13, 14].
[score:4]
It is worth noting that some N-deficiency-responsive miRNAs (e. g., miR169, miR172, miR394, miR395, miR397, miR398, miR399, miR827, miR408 and miR857) are also responsive to other nutrient stresses (i. e., B, P, Fe, S and Cu deficiencies) in plants [8, 10], indicating the involvement of miRNA -mediated crosstalk among N, B, P, Fe, S and Cu under N-deficiency.
[score:1]
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Other miRNAs from this paper: csi-MIR160a, csi-MIR169a, csi-MIR167c, csi-MIR167b, csi-MIR171b, csi-MIR171a, csi-MIR394a, csi-MIR399d, csi-MIR399a, csi-MIR399b, csi-MIR827, csi-MIR167a, csi-MIR160b, csi-MIR160c, csi-MIR167d, csi-MIR167e, csi-MIR169b, csi-MIR169c, csi-MIR169d, csi-MIR169e, csi-MIR169f, csi-MIR169g, csi-MIR169h, csi-MIR169i, csi-MIR169j, csi-MIR169k, csi-MIR169l, csi-MIR169m, csi-MIR169n, csi-MIR171c, csi-MIR171d, csi-MIR171e, csi-MIR171f, csi-MIR171g, csi-MIR171h, csi-MIR171i, csi-MIR394b, csi-MIR399e, csi-MIR399f, csi-MIR169o, csi-MIR169p, csi-MIR169q, csi-MIR169r
The roles of P-deficiency -induced up-regulation of plant miR399 and miR827 in the maintenance of P homeostasis via inhibiting their targets ubiquitin-conjugating enzyme E2 24 (UBC24) and N limitation adaptation (NLA), respectively have been well characterized [14, 23, 26, 27].
[score:6]
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Other miRNAs from this paper: csi-MIR164a, csi-MIR166e, csi-MIR169a, csi-MIR398a, csi-MIR166a, csi-MIR156a, csi-MIR3946, csi-MIR159a, csi-MIR166c, csi-MIR167c, csi-MIR167b, csi-MIR159d, csi-MIR395a, csi-MIR397, csi-MIR399d, csi-MIR399a, csi-MIR399b, csi-MIR408, csi-MIR482f, csi-MIR482a, csi-MIR482b, csi-MIR535a, csi-MIR827, csi-MIR857, csi-MIR482c, csi-MIR167a, csi-MIR156b, csi-MIR156c, csi-MIR156d, csi-MIR156e, csi-MIR156f, csi-MIR156g, csi-MIR159b, csi-MIR159c, csi-MIR164b, csi-MIR164c, csi-MIR166b, csi-MIR166d, csi-MIR166f, csi-MIR166g, csi-MIR166h, csi-MIR166i, csi-MIR166j, csi-MIR166k, csi-MIR167d, csi-MIR167e, csi-MIR169b, csi-MIR169c, csi-MIR169d, csi-MIR169e, csi-MIR169f, csi-MIR169g, csi-MIR169h, csi-MIR169i, csi-MIR169j, csi-MIR169k, csi-MIR169l, csi-MIR169m, csi-MIR169n, csi-MIR395b, csi-MIR395c, csi-MIR398b, csi-MIR399e, csi-MIR399f, csi-MIR482d, csi-MIR535b, csi-MIR535c, csi-MIR482e, csi-MIR482g, csi-MIR156h, csi-MIR156i, csi-MIR156j, csi-MIR164d, csi-MIR169o, csi-MIR169p, csi-MIR169q, csi-MIR535d, csi-MIR535e, csi-MIR535f, csi-MIR169r
Plant miR399 and miR827, which are specifically induced by phosphorus (P)-deficiency, play a role in the regulation of P homeostasis by down -regulating their target genes UBC24 and nitrogen (N) limitation adaptation (NLA), respectively (Shukla et al., 2008; Hsieh et al., 2009).
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