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28 publications mentioning ssc-mir-155

Open access articles that are associated with the species Sus scrofa and mention the gene name mir-155. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

[+] score: 78
Other miRNAs from this paper: ssc-mir-124a-2, ssc-mir-124a-1
Recently, mir-155 has been reported to target (inhibit) the other two significantly enriched gene expression regulators, SPI1 and CEBPB [93], [94], [95], thereby explaining the expression pattern for much of the sub-network shown in Figure 8. The RNA expression level changes, confirmed by qPCR for TLR4, CEBPB, SPI1 and miR-155, provide substantial evidence to support the SNEA-generated hypothesis of the coordinated involvement of these factors in the regulation of the global gene expression responses introduced by ST inoculation. [score:15]
Integrative analysis of miR-155 regulation with two important transcription factors in the porcine blood transcriptome response to Salmonella Beyond the IFN-γ regulon, we further explored selected regulators showing large numbers of target genes in the differentially expressed gene lists, including TLR4, CEBPB, and SPI1, all of which we confirmed by q-PCR as up-regulated genes. [score:10]
In addition, one of the miRNAs with significantly enriched targets within the up-regulated genes in PS1 animals is mir-155, which was down-regulated over 5-fold following ST challenge in persistent shedder pigs in a separate challenge population (PS3) (p<0.01). [score:9]
0028768.g007 Figure 7Reciprocal expression during the persistent shedder response to Salmonella inoculation for miR-155 and two of its repressed targets, SPI1 and CEBPB, which themselves have over-represented target gene sets in the transcriptomic response to Salmonella. [score:7]
Common gene interactions of the IFNG, SPI1, TLR4, CEBPB and mir-155 (highlighted with yellow) gene expression regulation networks for up-regulated genes in the PS animals. [score:7]
Reciprocal expression during the persistent shedder response to Salmonella inoculation for miR-155 and two of its repressed targets, SPI1 and CEBPB, which themselves have over-represented target gene sets in the transcriptomic response to Salmonella. [score:7]
The expression levels of two miRNAs (mir-124 and mir-155) with over-represented targets were quantified by Stem-loop TaqMan Assay (Figure 7). [score:4]
While the expression of mir-124 in peripheral blood was almost undetectable (data not shown), miR-155 was down regulated in pig peripheral blood RNA from PS3 animals inoculated with ST (over 5 fold, p<0.01, Figure 7). [score:4]
They demonstrated that lower levels of miR-155 increased IFNGR1 RNA levels and that the IFNGR1 mRNA had a functional miR-155 target sequence. [score:3]
Banerjee and colleagues showed that during CD4 [+] T cell activation miR-155 levels increase and promote Th1 differentiation, while suppression of miR-155 levels promoted Th2 differentiation. [score:3]
Our results add evidence of a direct connection between miR-155 and IFN-γ signaling [96]. [score:2]
Note the large number of genes in common among the IFNG, SPI1 and CEBPB regulons, as well as that miR-155 negatively regulates both SPI1 and CEBPB. [score:2]
Integrative analysis of miR-155 regulation with two important transcription factors in the porcine blood transcriptome response to Salmonella. [score:2]
Consistent with these regulatory interactions, we note that in PS1 animals (high IFN-γ low miR-155), IFNGR1 RNA was significantly increased after Salmonella inoculation (3.6 fold, q<0.002) while in LS1 animals the increase is less dramatic (1.8 fold, q<0.13). [score:2]
The miR155 data was collected only from LS3PS3 due to the fact that sufficient samples for the other pigs for miRNA preparations were unavailable. [score:1]
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[+] score: 42
As examples, the liver-specific miR-122 promotes the replication of hepatitis C virus (HCV) [17], [18], while miR-196, miR-199, miR-296, miR-351, miR-431 and miR-448 inhibit HCV genome propagation [19], [20]; miR-32 effectively restricts the accumulation of primate foamy virus type 1 (PFV-1) in human cells [21]; miR-323, miR-491 and miR-654 inhibit the replication of the H1N1 influenza A virus by binding to the viral PB1 gene [22]; miR-28, miR-125b, miR-150, miR-223 and miR-382 target the 3′ end of human immunodeficiency virus (HIV) mRNA, thereby restricting HIV production [23]; miR-199a-3p and miR-210 limit the hepatitis B virus (HBV) surface antigen and polymerase production by degrading and/or inhibiting translation of viral mRNAs encoding these proteins [24]; overexpression of miR-24 and miR-93 suppresses vesicular stomatitis virus (VSV) replication through targeting the viral genes encoding RNA -dependent RNA polymerase (L protein) and phosphoprotein (P protein), respectively [25]; in macrophages, upregulation of miR-155 suppresses VSV replication, while inhibition of miR-155 had the opposite effect. [score:24]
Interestingly, instead of directly acting on VSV RNA, miR-155 was shown to target the expression of SOCS1, a negative regulator of type I interferon signaling, thereby indirectly enhancing the anti-viral state of the cell [26]. [score:8]
We compared the basal expression levels of miR-125b, miR-155, miR-23a and miR-365 in PAMs by qPCR and found that miR-125b was among the most highly expressed miRNAs examined (data not shown). [score:4]
The mimics and inhibitors of miR-24, miR-93, miR-122, miR-125b, miR-146a, miR-155, miR-181, miR-196, miR-351, and miR-365 (shown in Table S1) were obtained from GenePharma (Shanghai, China). [score:3]
To screen the potential miRNAs which can reduce PRRSV replication, the mimics or inhibitors of 10 miRNAs (Table S1), including miR-24, miR-93, miR-122, miR-125b, miR-146a, miR-155, miR-181, miR-196, miR-351, and miR-365, were chosen and synthetized. [score:3]
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[+] score: 42
In our RT-qPCR study, miR-155 was significantly down-regulated under pathogen triggered inflammation and so was mRNA coding for the miR-155 target IFN-γ in the same animals [23]. [score:6]
MiR-155 has a slightly different profile with all the infected samples having significantly lower expression than the control sample but at the same time the expression does not differ significantly between samples representing various stages of infection (similar read count number confirms this expression pattern). [score:6]
Such dramatic disturbance in regulation of immune response could be the explanation for the surprisingly reversed miR-155 expression profile. [score:4]
Moreover, miR-155 has been shown to be induced by the bacterium Helicobacter pylori, a known pathogen of the human stomach [19] and has been proven to act as a global immune regulator in endotoxin-tolerant macrophages, proving that microRNA expression depends strongly on the status of infected cells [20]. [score:4]
LPS stimulation results in up regulation of miR-155 expression. [score:4]
Group 2: microRNAs down-regulated in necrotic comparing to visually unaffected areas and/or control sample (miR-126, miR-155). [score:4]
The miRNA candidates assayed by RT-qPCR are found to target multiple mRNAs as shown in Table  6. Two out of 13 microRNAs (miR-15a, miR-155) included in Table  6 are predicted to target more than 20 different mRNAs. [score:4]
MiR-155, a known key player in inflammation, was found expressed in both samples. [score:2]
Literature reports a number of microRNAs, mainly miR-21, miR-155, miR-146a, miR-223 to be important regulators of the protein coding genes involved in the above mentioned pathways [60], [61], [63]. [score:2]
Most of the miRNA candidates are falling into highly abundant or up/down regulated microRNAs in the necrotic sample, however the two miRNAs: miR-15a and miR-155 were chosen due to their biological relevance as reported in the literature disregarding their rather low read count (). [score:2]
For example, miR-146a/b and miR-155 have been induced by TLR4 -mediated sensing of bacterial lipopolysaccharide (LPS) [16], [17]. [score:1]
The following key player in control of immunity and inflammation, miR-155, showed rather contradicting results to those existing in the literature [18]; [67]; [68]. [score:1]
Of the miRNAs investigated in the present study, miR15a, miR21, miR126, miR142-5p, miR144-5p, miR146a-5p, miR148a, miR152, miR155, miR192, miR-223, miR-45 and miR-d5 are 5'-miRNAs hence only the mature miRNAs of these targets were detected. [score:1]
The ncRNAs chosen for RT-qPCR validation were: miR-15a, miR-21, miR-126, miR-142-5p, miR-143-3p, miR-144*, miR-146a-5p, miR-148a, miR-155, miR-223, miR-451, miR-664-5p, miR-d5 and SNORD15. [score:1]
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[+] score: 22
One study demonstrated that miR-155 might target the chicken anti-influenza gene MX1 and activate the JUK pathway, therefore regulating influenza virus infection in chickens [22]. [score:4]
It was important to note that except for mmu-miR-155 and mmu-miR-142-3p, the subset of differentially expressed microRNAs on 2 dpi was distinct from that on 5 dpi. [score:3]
Notably, mmu-miR-155 was commonly differentially expressed between both H1N1 strains at all time points. [score:3]
In the present study, we successfully profiled the lung cellular microRNAs of mice infected with the 2009 pandemic influenza virus BJ501 and a comparison influenza virus PR8, and 29 microRNAs were found to be differentially expressed in response to influenza virus BJ501 infection compared to 43 to PR8; among them, 15 had no reported function in Pubmed, while 32 including miR-145, miR-155 and miR-233 were known to associate with cancer, immunity and antiviral activities. [score:2]
miR-155 knockout mice cannot generate defensive immune responses as a result of affected lymphocyte differentiation [36]. [score:2]
A group of microRNAs including miR-155 and miR-223 have been identified to potentially regulate influenza virus infection in our study. [score:2]
Although this study generated a list of candidate microRNAs including miR-155 and miR-223 that potentially regulate influenza virus infections, additional studies are warranted to clarify the mechanisms behind how these candidate microRNAs mediate host–virus interactions during influenza virus infection. [score:2]
Nine microRNAs (miR-1, miR-1187, miR-133a, miR-133b, miR-155, miR-2137, miR-223, miR-30d and miR-574-3p) were selected for validation. [score:1]
Thus, it is plausible to assume that the miR-155 and miR-223 may play an important role in the response to influenza virus infection. [score:1]
miR-155 has been demonstrated to play an important role in the mammalian immune system. [score:1]
In our study, miR-155 and miR-223 were significantly induced by both the 2009 pandemic H1N1 virus and the PR8 virus. [score:1]
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[+] score: 19
SPI1 is a known target of miR-155 13 and thus the down-regulation of miR-155 is consistent with the increased expression of SPI1 after Salmonella challenge. [score:8]
This approach identified several known miRNA-mRNA interactions, for example, SPI1 and SOCS1 as the targets of miR-155 (regulatory effects −0.32 and −0.30), and BAK1 as the target of miR-125a (regulatory effect −0.36). [score:7]
For example, miR-24, miR-146a, miR-155 and miR-214 were predicted to be involved in regulation of the Toll-like receptor (TLR) signaling pathway (Table 3). [score:2]
Several miRNA hub nodes were identified including miR-146a, miR-155, miR-214 and miR-331-3p (Fig. 3). [score:1]
It has been shown that miR-146a and miR-155 are involved in the TLR signaling pathway and play important roles in innate immune response 13. [score:1]
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[+] score: 18
The expression level of miR-155 was also found to be increased during host antiviral responses; for example, it was significantly upregulated in EBV-infected lymphocytes and interfered with the balance of gene expression by modulating transcriptional regulatory factors [41– 45]. [score:9]
Meanwhile, miR-155 was found to be significantly upregulated in PCMV-infected porcine macrophages and porcine parvovirus-infected PK-15 cells (Additional file  1: Table S5). [score:4]
miR-155-5p was also upregulated in PCMV-infected lung (526% increase), liver (239% increase) and spleen (493% increase) compared with that in the uninfected control. [score:3]
Faraoni I Antonetti FR Cardone J Bonmassar E miR-155 gene: a typical multifunctional microRNABiochim. [score:1]
In addition, a previous study showed that miR-155 plays an important role in antimicrobial infection. [score:1]
[1 to 20 of 5 sentences]
[+] score: 17
Interestingly, miR-155 is highly upregulated following ischaemic or toxic injury to the kidneys [27, 28]. [score:4]
In this study, we show for the first time that there is a relationship between miR-155 expression and immune and metabolic parameters in healthy animals. [score:3]
Overexpression of liver miR-155 in transgenic mice resulted in significantly reduced levels of serum total cholesterol, triglycerides (TG) and high-density lipoprotein (HDL) [26]. [score:3]
We found that expression levels of miR-155 were highly negatively correlated with TG, ALB, BUN and IP. [score:3]
In fact, miR-155 plays a crucial role in several physiological processes including haematopoietic lineage differentiation, immunity and inflammation [23, 24]. [score:1]
At the same time, miR-155 was also correlated with erythrocyte levels (RBC, HGB, HCT and MCHC) and LYM. [score:1]
miR-155 is also involved in adipogenesis and lipid metabolism [25, 26]. [score:1]
For example, serum TG was correlated with miR-744, miR-199, miR-10, miR-23, miR-195 and miR-155, which were also correlated with erythrocyte-related traits (RBC, MCHC, HGB and MCV). [score:1]
[1 to 20 of 8 sentences]
[+] score: 17
Liu S., Yang Y., and Wu J. R. 2011 TNF alpha -induced up-regulation of miR-155 inhibits adipogenesis by down -regulating early adipogenic transcription factors. [score:7]
Liu and et al. (52) found that TNF-α treatment led to the upregulation of miR-155 through the nuclear factor κB (NFκB) pathway in 3T3-L1 cells, and miR-155 targets 3′-UTRs of C/EBPβ and cAMP response element binding protein(CREB). [score:6]
Li H., Xie S., Liu M., Chen Z., Liu X., Wang L., Li D., and Zhou Y. 2014 The clinical significance of downregulation of mir-124–3p, mir-146a-5p, mir-155–5p and mir-335–5p in gastric cancer tumorigenesis. [score:4]
[1 to 20 of 3 sentences]
[+] score: 17
However, another study revealed that miR-155 was significantly down-regulated in circulation of the CHD patients [26], and acted as an anti-inflammatory and atheroprotective miRNA, by inhibiting atherosclerotic plaque development, maintaining plaque stability, and decreasing monocyte subset differentiation [27]. [score:7]
Recently, miR-155-5p was reported to be upregulated after plaque rupture for 0.5 h and 1 h [25], and miR-124, highly expressed in monocyte/macrophage cells, was as well a promising biomarker for AMI diagnosis [10, 19], suggesting that monocytes-released miRNAs could be useful for AMI early diagnosis. [score:6]
MiR-155 was reported to be upregulated in human carotid plaque, and could prompt the inflammatory response during atherosclerosis by repressing B-cell leukemia/lymphoma 6 (Bcl6) in macrophages [25]. [score:3]
MiR-155, enriched in CD14 [+] monocytes, was closely related with inflammation and could pleiotropically regulate atherosclerosis [24]. [score:1]
[1 to 20 of 4 sentences]
[+] score: 14
GO annotation was performed for the target genes of five designated differentially expressed miRNAs (miR-146b, miR-155–5p, miR-195, miR-124a and miR-1306–5p). [score:5]
In this study, the sequencing data confirmed the results of previous studies, and proved that in addition to immune cells, normal cells also abundantly expressed miR-146 and miR-155 when stimulated by viral antigens and are involved in the regulation of immunity. [score:4]
In particular, mature miR-146 and miR-155 were significantly upregulated in infected ST cells compared with the control group; the fold changes of miR-146 and miR-155 between the infected ST cells and the control group were 3.30 and 1.74, respectively. [score:3]
Previous research showed that miR-146 and miR-155 play a key regulatory role on host immune function. [score:2]
[1 to 20 of 4 sentences]
[+] score: 13
The summary of the six DE miRNA common to all species is described in Fig.   5. Briefly, all DE candidates were single copy miRNA across all libraries, and 4 DE miRNA (miR-101-3p, miR-16-5p, miR-143-3p and miR-155-5p) were up-regulated in bats while 2 (miR-125-5p and miR-221-5p) were down-regulated. [score:7]
Interestingly, miR-155 also targets Bach1, a negative regulator of Nrf2, which is a core activator of the antioxidant response signaling pathway [38]. [score:4]
miR-155 is a multifunctional miRNA regulating inflammation, immunity and oncogenic pathways [37]. [score:2]
[1 to 20 of 3 sentences]
[+] score: 13
Among them, let-7 g, miR-17-5p, miR-17-3p, miR-20a, miR-181a, miR-16, miR-146b, miR-10b, and miR-155-5p were upregulated; let-7c, miR-122, miR-18a, miR-19a, miR-19b, miR-196b, miR-21, and miR-9 were downregulated. [score:7]
However, we did not detect differential expression of other previously identified miRNAs (miR-223, miR-150, miR-92a), although miR-10b, miR-20a, miR-30a-5p, miR-34a, miR-17—5p, miR-16, miR-146b, and miR-155-5p expression was significantly different. [score:5]
Many immune-related miRNAs have been identified in innate and adaptive immune systems, including the miR-17—92 cluster, miR-221, miR-10, miR-196b, miR-126, miR-155, miR-150; miR-181a, miR-326, miR-142-3p, miR-424, miR-21, miR-106a, miR-223, miR-146; the let-7 family, miR-9, and miR-34 [6]. [score:1]
[1 to 20 of 3 sentences]
[+] score: 13
Only 3 miRNAs (miR-155, miR-150-1 and miR-204) at W1 and W4, and 4 miRNAs (miR-132, miR-146b, miR-212 and miR-218-2) at W4 and W7 were consistently upregulated across two of three time points. [score:4]
In our study, other 6 miRNAs (miR-155, miR-150-1, miR-204, miR-132, miR-212 and miR-218-2) were consistently upregulated across two of three weaning times. [score:4]
miR-155 was earlier found to play critical roles in various physiological and pathological processes such as immunity, inflammation, cancer, cardiovascular disease and haematopoietic lineage differentiation, and involved in viral infections [50]. [score:3]
The mechanisms of miR-155 on host inflammation and immunity were demonstrated to be associated with regulating the TLR3/TLR4 (toll-like receptors) pathways and responsiveness to interferon [51], [52]. [score:2]
[1 to 20 of 4 sentences]
[+] score: 11
We found that EGFR was regulated by miR-342, miR-155, miR-30b, miR-210, miR-192, let-7g, and miR-146b-5p, which were all down expressed in H1N1 critically ill patients. [score:4]
validation of differentially expressed miRNAs and ROC analysisThe microarray data were validated by performing, qRT-PCR for nine miRNAs, including hsa-miR-146b-5p, hsa-miR-148a, hsa-miR-150, hsa-miR-31, hsa-miR-155, hsa-miR-29a, hsa-miR-29b, hsa-miR-342-5p, and hsa-miR-886-3p. [score:3]
The expression of hsa-miR-150, hsa-miR-31, hsa-miR-155, hsa-miR-29a, hsa-miR-29b, hsa-miR-342-5p, and hsa-miR-146b-5p were present in lower abundance, whereas hsa-miR-148a and hsa-miR-886-3p were present in higher abundance in PBMCs from critically ill patients infected with H1N1 influenza virus than that from healthy controls. [score:3]
The microarray data were validated by performing, qRT-PCR for nine miRNAs, including hsa-miR-146b-5p, hsa-miR-148a, hsa-miR-150, hsa-miR-31, hsa-miR-155, hsa-miR-29a, hsa-miR-29b, hsa-miR-342-5p, and hsa-miR-886-3p. [score:1]
[1 to 20 of 4 sentences]
[+] score: 10
The miR-155 can target the TLR/IL-1 inflammatory pathway and serves as a negative feedback loop to modulate inflammatory cytokine production in response to microbial stimulation [12]. [score:3]
Recent studies have showed that miR-155 is differentially expressed in pig during Salmonella Typhimurium infection [13]. [score:3]
In human monocytes, the miR-146 and miR-155 can respond to endotoxins and a variety of microbial components and pro-inflammatory cytokines [9]. [score:1]
In addition to miR-146 and miR-155, the let-7, miR-15, miR-128 and miR-29a also have roles in Salmonella infection. [score:1]
Analysis of host miRNA by high throughput sequencing confirmed the co-induction of miR-146 and miR-155 upon live microbial infection with wild type (WT) Salmonella Typhimurium strains [10]. [score:1]
Previous studies found that miR-146 and miR-155 were co -induced in macrophage cells in response to microbial LPS [7– 9]. [score:1]
[1 to 20 of 6 sentences]
[+] score: 8
We also found only low and unchanged expression of the microRNAs, ssc-miR-155-5p, ssc-miR-181b and ssc-miR-191, even though they have previously implicated in CRC development in humans [31– 33]. [score:4]
Ten differentially expressed miRNAs were validated by qRT-PCR: ssc-let-7e, ssc-miR-98, ssc-miR-126-3p, ssc-miR-146a-5p, ssc-miR-146b, ssc-miR-155-5p, ssc-miR-181b, ssc-miR-183, ssc-miR-191 and ssc-miR-196a. [score:3]
Differences in ssc-miR-155-5p, ssc-miR-181b and ssc-miR-191 levels fell below statistical significance, but were still consistent with data from small RNA sequencing (Figure 2). [score:1]
[1 to 20 of 3 sentences]
[+] score: 5
Other miRNAs from this paper: mmu-mir-155
In this research, five artificial microRNAs (amiRNAs) respectively targeted towards ORF5 (amirGP5-243, -370) and ORF6 (amirM-82, -217,-263) were designed and incorporated into a miRNA -based vector that mimics the backbone of murine miR-155 and permits high expression of amiRNAs in a GFP fused form mediated by RNA Pol II promoter CMV. [score:5]
[1 to 20 of 1 sentences]
[+] score: 4
MiR-155 can regulate the expression of Olfactomedin-like 3 (OLFML3), which may affect porcine prenatal skeletal muscle development [10]. [score:4]
[1 to 20 of 1 sentences]
[+] score: 4
The miR-155 plays a role in prenatal skeletal muscle development by mediating the expression of the olfactomedin-like 3 (OLFML3) gene in pigs [18]. [score:4]
[1 to 20 of 1 sentences]
[+] score: 4
S25252) miR-30c-5p Potential regulator of EIF2A 63 2.14E-19 −7.805 Jiang et al. 2016 (10.1038/srep21565) miR-155-5p Potential regulator of DNMT1 73 1.24E-15 −8.437 Zhang et al. 2015 (10.1093/nar/gkv518) miR-483-3p Potential regulator of CDC25A 25 2.50E-08 −4.969 Bertero et al. 2013 (10.1038/cdd. [score:4]
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[+] score: 3
miR-155-5P, miR-221-5P, let-7f, and miR-181c-1 were either absent or not detectable in the samples. [score:1]
miR-155-5P, miR-221-5P, let-7f, and miR-181c-1 were either absent or not detectable in our samples (Fig. 4d). [score:1]
However, a few miRNAs such as miR-155, miR-221, let-7f, miR-181C-1 were not detected in the EVs released by PAOEC and we are unable to speculate about their absence. [score:1]
[1 to 20 of 3 sentences]
[+] score: 3
Many studies have demonstrated that miRNA expression profiles are subject to change in different cells when stimulated by LPS via TLR-signaling pathways, including miR-146a, miR-155, miR-132, miR-15a/16, miR-27a and miR-532-5p [19, 20, 21, 22, 23]. [score:3]
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[+] score: 3
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-15a, hsa-mir-16-1, hsa-mir-21, hsa-mir-23a, hsa-mir-24-1, hsa-mir-24-2, hsa-mir-26a-1, hsa-mir-29a, hsa-mir-30a, hsa-mir-31, hsa-mir-99a, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-103a-2, hsa-mir-103a-1, hsa-mir-16-2, hsa-mir-192, hsa-mir-148a, hsa-mir-10b, hsa-mir-181a-2, hsa-mir-181a-1, hsa-mir-215, hsa-mir-223, hsa-mir-224, hsa-mir-200b, hsa-mir-15b, hsa-mir-27b, hsa-mir-125b-1, hsa-mir-141, hsa-mir-143, hsa-mir-152, hsa-mir-125b-2, hsa-mir-126, hsa-mir-146a, hsa-mir-184, hsa-mir-200c, hsa-mir-155, hsa-mir-29c, hsa-mir-200a, hsa-mir-99b, hsa-mir-296, hsa-mir-30e, hsa-mir-26a-2, hsa-mir-378a, hsa-mir-342, hsa-mir-148b, hsa-mir-451a, ssc-mir-125b-2, ssc-mir-148a, ssc-mir-15b, ssc-mir-184, ssc-mir-224, ssc-mir-23a, ssc-mir-24-1, ssc-mir-26a, ssc-mir-29b-1, ssc-let-7f-1, ssc-mir-103-1, ssc-mir-21, ssc-mir-29c, hsa-mir-486-1, hsa-mir-499a, hsa-mir-671, hsa-mir-378d-2, bta-mir-26a-2, bta-mir-29a, bta-let-7f-2, bta-mir-103-1, bta-mir-148a, bta-mir-16b, bta-mir-21, bta-mir-499, bta-mir-99a, bta-mir-125b-1, bta-mir-126, bta-mir-181a-2, bta-mir-27b, bta-mir-31, bta-mir-15b, bta-mir-215, bta-mir-30e, bta-mir-148b, bta-mir-192, bta-mir-200a, bta-mir-200c, bta-mir-23a, bta-mir-29b-2, bta-mir-29c, bta-mir-10b, bta-mir-24-2, bta-mir-30a, bta-mir-200b, bta-let-7a-1, bta-mir-342, bta-let-7f-1, bta-let-7a-2, bta-let-7a-3, bta-mir-103-2, bta-mir-125b-2, bta-mir-15a, bta-mir-99b, hsa-mir-664a, ssc-mir-99b, hsa-mir-103b-1, hsa-mir-103b-2, ssc-mir-15a, ssc-mir-16-2, ssc-mir-16-1, bta-mir-141, bta-mir-143, bta-mir-146a, bta-mir-152, bta-mir-155, bta-mir-16a, bta-mir-184, bta-mir-24-1, bta-mir-223, bta-mir-224, bta-mir-26a-1, bta-mir-296, bta-mir-29d, bta-mir-378-1, bta-mir-451, bta-mir-486, bta-mir-671, bta-mir-29e, bta-mir-29b-1, bta-mir-181a-1, ssc-mir-181a-1, ssc-mir-215, ssc-mir-30a, bta-mir-2318, bta-mir-2339, bta-mir-2430, bta-mir-664a, bta-mir-378-2, ssc-let-7a-1, ssc-mir-378-1, ssc-mir-29a, ssc-mir-30e, ssc-mir-499, ssc-mir-143, ssc-mir-10b, ssc-mir-486-1, ssc-mir-152, ssc-mir-103-2, ssc-mir-181a-2, ssc-mir-27b, ssc-mir-24-2, ssc-mir-99a, ssc-mir-148b, ssc-mir-664, ssc-mir-192, ssc-mir-342, ssc-mir-125b-1, oar-mir-21, oar-mir-29a, oar-mir-125b, oar-mir-181a-1, hsa-mir-378b, hsa-mir-378c, ssc-mir-296, ssc-mir-146a, bta-mir-148c, ssc-mir-126, ssc-mir-378-2, ssc-mir-451, hsa-mir-378d-1, hsa-mir-378e, hsa-mir-378f, hsa-mir-378g, hsa-mir-378h, hsa-mir-378i, hsa-mir-451b, hsa-mir-499b, ssc-let-7a-2, ssc-mir-486-2, hsa-mir-664b, hsa-mir-378j, ssc-let-7f-2, ssc-mir-29b-2, ssc-mir-31, ssc-mir-671, bta-mir-378b, bta-mir-378c, hsa-mir-486-2, oar-let-7a, oar-let-7f, oar-mir-103, oar-mir-10b, oar-mir-143, oar-mir-148a, oar-mir-152, oar-mir-16b, oar-mir-181a-2, oar-mir-200a, oar-mir-200b, oar-mir-200c, oar-mir-23a, oar-mir-26a, oar-mir-29b-1, oar-mir-30a, oar-mir-99a, bta-mir-664b, chi-let-7a, chi-let-7f, chi-mir-103, chi-mir-10b, chi-mir-125b, chi-mir-126, chi-mir-141, chi-mir-143, chi-mir-146a, chi-mir-148a, chi-mir-148b, chi-mir-155, chi-mir-15a, chi-mir-15b, chi-mir-16a, chi-mir-16b, chi-mir-184, chi-mir-192, chi-mir-200a, chi-mir-200b, chi-mir-200c, chi-mir-215, chi-mir-21, chi-mir-223, chi-mir-224, chi-mir-2318, chi-mir-23a, chi-mir-24, chi-mir-26a, chi-mir-27b, chi-mir-296, chi-mir-29a, chi-mir-29b, chi-mir-29c, chi-mir-30a, chi-mir-30e, chi-mir-342, chi-mir-378, chi-mir-451, chi-mir-499, chi-mir-671, chi-mir-99a, chi-mir-99b, bta-mir-378d, ssc-mir-378b, oar-mir-29b-2, ssc-mir-141, ssc-mir-200b, ssc-mir-223, bta-mir-148d
A differential expression of four immune related miRNAs, miR-125b, miR-155, miR-146a, and miR-223 upon stimulation of bovine monocytes with LPS or Staphylococcus aureus enterotoxin B was demonstrated (Dilda et al., 2012). [score:3]
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[+] score: 1
Other miRNAs from this paper: hsa-mir-223, hsa-mir-142, hsa-mir-155, ssc-mir-142, ssc-mir-223
For example, levels of miR-142-5p, miR-155, and miR-223 can each predict acute rejection with >90% sensitivity and specificity in human renal allografts [94, 95]. [score:1]
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[+] score: 1
Other miRNAs from this paper: ssc-mir-30d
We identified one lncRNA TCONS_00056284, as a potential pre-miRNA of miR-155 (Supplementary Table  S7), which promotes type I IFN signalling in the antiviral innate immune response [37]. [score:1]
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[+] score: 1
Other miRNAs from this paper: hsa-mir-17, hsa-mir-28, hsa-mir-223, hsa-mir-127, hsa-mir-188, hsa-mir-194-1, hsa-mir-155, hsa-mir-194-2, hsa-mir-30e, hsa-mir-362, hsa-mir-363, hsa-mir-367, hsa-mir-379, hsa-mir-196b, hsa-mir-450a-1, hsa-mir-431, ssc-mir-28, hsa-mir-493, hsa-mir-512-1, hsa-mir-512-2, hsa-mir-500a, hsa-mir-501, hsa-mir-502, hsa-mir-450a-2, hsa-mir-513a-1, hsa-mir-513a-2, hsa-mir-506, hsa-mir-508, hsa-mir-509-1, hsa-mir-532, hsa-mir-615, hsa-mir-660, bta-mir-127, bta-mir-30e, bta-mir-17, bta-mir-450a-2, bta-mir-532, bta-mir-363, bta-mir-660, hsa-mir-891a, hsa-mir-892a, hsa-mir-509-2, hsa-mir-450b, hsa-mir-892b, hsa-mir-708, hsa-mir-509-3, hsa-mir-1285-1, hsa-mir-1285-2, hsa-mir-1248, ssc-mir-17, bta-mir-155, bta-mir-188, bta-mir-194-2, bta-mir-196b, bta-mir-223, bta-mir-28, bta-mir-362, bta-mir-367, bta-mir-379, bta-mir-431, bta-mir-493, bta-mir-500, bta-mir-502a-1, bta-mir-502a-2, bta-mir-502b, bta-mir-615, bta-mir-708, bta-mir-1248-1, bta-mir-1248-2, ssc-mir-450a, bta-mir-2320, bta-mir-1388, bta-mir-194-1, bta-mir-450a-1, eca-mir-30e, eca-mir-367, eca-mir-684, eca-mir-196b, eca-mir-615, eca-mir-708, eca-mir-194-1, eca-mir-493a, eca-mir-17, eca-mir-1248, eca-mir-28, eca-mir-127, eca-mir-379, eca-mir-431, eca-mir-493b, eca-mir-155, eca-mir-194-2, eca-mir-188, eca-mir-223, eca-mir-362, eca-mir-363, eca-mir-450a, eca-mir-450b, eca-mir-450c, eca-mir-500-1, eca-mir-500-2, eca-mir-501, eca-mir-502, eca-mir-508, eca-mir-509a, eca-mir-532, eca-mir-660, ssc-mir-30e, ssc-mir-196b-1, ssc-mir-450b, ssc-mir-127, ssc-mir-532, ssc-mir-708, ssc-mir-1285, ssc-mir-500, hsa-mir-514b, ssc-mir-363-1, ssc-mir-450c, hsa-mir-500b, ssc-mir-194b, ssc-mir-362, bta-mir-3601, ssc-mir-615, ssc-mir-2320, bta-mir-450b, ssc-mir-194a, ssc-mir-196b-2, ssc-mir-363-2, ssc-mir-493, hsa-mir-892c, eca-mir-1388, eca-mir-514b, eca-mir-506a, eca-mir-509b, bta-mir-194b, ssc-mir-1388, ssc-mir-223, ssc-mir-660, bta-mir-194b-2, bta-mir-1949
Sometimes these duplicated annotations were found twice on the same chromosome in close proximity (for example mir-196b twice on the same strand, and both mir-615 and mir-194 twice, once on each strand) and sometimes they were found both on the assembled chromosomes and within the unplaced contigs (for example mir-127, mir-155). [score:1]
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A) ssc-miR-215, B) MDM238, C) MDM392, D) ssc-miR-30a, E) ssc-miR-194, F) ssc-miR-374b, G) ssc-miR-155, H) ssc-miR-4331. [score:1]
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[+] score: 1
Other miRNAs from this paper: mmu-mir-155, hsa-mir-155
Each oligonucleotide also contained five nucleotides (TGCTG) derived from the endogenous miR-155 at the 5′ end and 19 nucleotides derived from miR-155 to form a terminal loop. [score:1]
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