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31 publications mentioning hsa-mir-877

Open access articles that are associated with the species Homo sapiens and mention the gene name mir-877. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

1
[+] score: 23
MicroRNA mimics and hairpin inhibitors for microRNA-574-5p (hsa-miR-574-5p), microRNA-656 (hsa-miR-656), microRNA-877-5p (hsa-miR-877-5p), and microRNA-921 (hsa-miR-921), as well as mimic and inhibitor negative controls, were obtained from Dharmacon (Chicago, IL). [score:5]
G/G/A/A Bioinformatic predictions miR-574-5p miR-877-5p miR-921 miR-656 CHO cells 35% ↓/52% ↓ NC/NC 46% ↓/NC N/A/NC Lymphoblastoid protein 48% ↓/49% ↓ NC/NC 40% ↓/NC N/A/NC Lymphoblastoid activity 54% ↓/56% ↓ NC/NC 18% ↓/NC N/A/NC Although the SNP rs9024 does not directly alter the 3′-UTR target binding sites of hsa-miR-574-5p (position 120) or hsa-miR-921 (position 125) seed regions, it is notable that both binding sites are proximal to the polymorphic site at position 133 (Figure 1). [score:4]
Cells were co -transfected with 15 nM miRNA mimic (hsa-miR-574-5p, hsa-miR-656, hsa-miR-877, or hsa-miR-921) and miRNA mimic negative control in the presence or absence of 5 nM of specific miRNA inhibitor using the Neon transfection system (Invitrogen). [score:3]
CHO-K1 cells were co -transfected with CBR1 3′-UTR luciferase reporter constructs (500 ng) plus internal control plasmid pRL-TK (50 ng) and 15 nM miRNA mimic (hsa-miR-574-5p, hsa-miR-656, hsa-miR-877-5p, or hsa-miR-921) in the presence or absence of 5 nM specific miRNA inhibitor using DharmaFECT Duo transfection reagent (Dharmacon). [score:3]
Free energy predictions from the PITA algorithm microRNA ▵G [duplex] ▵G [open] ▵▵G G A G A G A hsa-miR-574-5p −24.49 −24.49 −6.54 −5.41 −17.94 −19.07 hsa-miR-877-5p −19.4 −19.4 −12.75 −13.33 −6.64 −6.06 hsa-miR-921 −11.6 −5.8 −5.92 −8.04 −5.67 2.24 hsa-miR-656 N/A −10 N/A −5.05 N/A −4.94 ▵G values expressed in kcal/mol Potential miRNA binding sites on the 3′-UTR of CBR1 and their relative locations are indicated by shaded regions. [score:3]
Consistent with their lack of interaction with CBR1 3′-UTR reporters in CHO cells, miR-656 and miR-877 failed to induce significant changes in CBR1 protein levels in lymphoblastoid cells homozygous for either rs9024 G or A alleles (Figure 3A and 3B). [score:1]
Additionally, hsa-miR-656 or hsa-miR-877-5p failed to significantly alter luciferase activity when paired with the pCBR1-1096A construct (Figure 2A). [score:1]
Meanwhile, transfection of hsa-miR-656 or hsa-miR-877-5p resulted in no change in luciferase activity. [score:1]
Bioinformatic predictions led us to examine hsa-miR-574-5p, hsa-miR-656, hsa-miR-877-5p, and hsa-miR-921 as prime CBR1 binding candidates. [score:1]
The algorithms we used pinpointed hsa-miR-574-5p, hsa-miR-656, hsa-miR-877-5p, and hsa-miR-921 as the most viable candidates for interacting with the 3′-UTR of CBR1 (Figure 1). [score:1]
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2
[+] score: 21
Among the 63 upregulated and 49 downregulated miRNAs in both the CP and CLP plasma samples, six miRNAs, namely miR-340–5p, miR-877–5p, miR-3648, miR-1260a, miR-494–3p, and miR-1304–3p, were selected for expression validation (Table 3). [score:9]
Relative expression level (2 [−ΔΔCt]) of miR-340-5p, miR-877-5p, miR-3648, miR-1260a, miR-494-3p, and miR-1304-3p expression in the plasma of cleft palate patients (n =16), cleft lip with cleft palate patients (n = 33) and controls (n = 8) (Mann-Whitney U test). [score:5]
The results showed that three miRNAs, miR-340–5p, miR-877–5p and miR-3648, were significantly upregulated in both the CP and CLP plasma samples (Figure 2). [score:4]
Six miRNAs, namely miR-340–5p, miR-877–5p, miR-3648, miR-1260a, miR-494–3p, and miR-1304–3p, were found to be differentially expressed in both the NSCP and NSCLP plasma samples. [score:3]
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3
[+] score: 16
In DLB, we detected coincident gene expression regulations with sCJD for miRNA-877-5p and miRNA-323a-5p (both with decreased expression levels) (Fig 6C). [score:6]
Among these, the deregulated expression levels of miRNA-195-5p, miRNA-877-5p and miRNA-323a-5p (marked in red) were previously validated by qPCR in the FC of sCJD cases (Fig 2). [score:4]
In AD samples, we detected coincident gene expression regulations with sCJD for miRNA-195-5p (increased) and for miRNA-877-5p and 323a-5p (decreased) (Fig 6B). [score:4]
There is virtually no information on the functions of the two commonly regulated miRNAs in the brain of the three dementias (miRNA-877-5p and miRNA-323a-5p). [score:2]
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4
[+] score: 15
133 genes were found as the target genes of hsa-miR-27b-3p and 11 genes were found as the target genes of hsa-miR-877-5p; however 70 genes were found as the target genes of hsa-miR-944. [score:7]
However the target genes of two miRNAs in TargetScan are CCM2, while more than two miRNAs' target genes are CCM2 in the nother two databases, both of them including hsa-miR-27b-3p, hsa-miR-877-5p, and hsa-miR-944 (Supplementary Table S5). [score:7]
According to the combination analysis, three miRNAs were found in the intersection, including hsa-miR-27b-3p, hsa-miR-944, and has-miR-877-5p. [score:1]
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5
[+] score: 12
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-21, hsa-mir-22, hsa-mir-23a, hsa-mir-24-1, hsa-mir-24-2, hsa-mir-25, hsa-mir-26b, hsa-mir-27a, hsa-mir-31, hsa-mir-33a, hsa-mir-99a, hsa-mir-100, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-103a-2, hsa-mir-103a-1, hsa-mir-199a-1, hsa-mir-148a, hsa-mir-147a, hsa-mir-34a, hsa-mir-182, hsa-mir-199a-2, hsa-mir-212, hsa-mir-221, hsa-mir-224, hsa-let-7g, hsa-let-7i, hsa-mir-27b, hsa-mir-30b, hsa-mir-125b-1, hsa-mir-130a, hsa-mir-132, hsa-mir-142, hsa-mir-145, hsa-mir-152, hsa-mir-153-1, hsa-mir-153-2, hsa-mir-125a, hsa-mir-125b-2, hsa-mir-127, hsa-mir-134, hsa-mir-200c, hsa-mir-106b, hsa-mir-361, hsa-mir-148b, hsa-mir-20b, hsa-mir-410, hsa-mir-202, hsa-mir-503, hsa-mir-33b, hsa-mir-643, hsa-mir-659, bta-let-7f-2, bta-mir-103-1, bta-mir-148a, bta-mir-21, bta-mir-221, bta-mir-26b, bta-mir-27a, bta-mir-99a, bta-mir-125a, bta-mir-125b-1, bta-mir-145, bta-mir-199a-1, bta-mir-27b, bta-mir-30b, bta-mir-31, bta-mir-127, bta-mir-142, bta-mir-20b, bta-let-7d, bta-mir-132, bta-mir-148b, bta-mir-200c, bta-mir-22, bta-mir-23a, bta-mir-29b-2, bta-mir-361, bta-let-7g, bta-mir-24-2, bta-let-7a-1, bta-let-7f-1, bta-let-7i, bta-mir-25, bta-let-7a-2, bta-let-7a-3, bta-let-7b, bta-let-7c, bta-let-7e, bta-mir-103-2, bta-mir-125b-2, bta-mir-34a, hsa-mir-708, hsa-mir-147b, hsa-mir-940, hsa-mir-548j, hsa-mir-302e, hsa-mir-103b-1, hsa-mir-103b-2, bta-mir-100, bta-mir-106b, bta-mir-130a, bta-mir-134, bta-mir-147, bta-mir-152, bta-mir-153-1, bta-mir-153-2, bta-mir-182, bta-mir-24-1, bta-mir-199a-2, bta-mir-202, bta-mir-212, bta-mir-224, bta-mir-33a, bta-mir-33b, bta-mir-410, bta-mir-708, bta-mir-877, bta-mir-940, bta-mir-29b-1, bta-mir-148c, bta-mir-503, bta-mir-148d
For this a total of six miRNAs representing those whose expression was induced (miR-212, miR-182 & let-7 g) or suppressed (miR-100, miR-877 and miR-200c) due to hyperstimulation were selected for detecting their expression in exosome and Ago2 fractions. [score:7]
On the other hand, miR-100 and miR-877 showed the greatest fold change regulation among the downregulated miRNAs (Table  1). [score:5]
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6
[+] score: 10
In addition to the two snoRNAs, RNU44 and RNU24, four miRNAs were also uniformly expressed in the two groups, namely miR-425* (P = 0.49; FC = 1.42), miR-877 (P = 0.35; FC = 1.39), miR-130b* (P = 0.91, FC = −1.13) and miR-181a-2* (P = 0.97; FC = 1.48). [score:3]
With the use of suitable EC reference genes, including hsa-miR-425*, RNU24, hsa-miR-877 and EC reference pair, RNU24/hsa-miR-425*, over -expression of all three miRNAs (hsa-miR-21*, hsa-miR-144* and hsa-miR-923) was identified, as previously reported [47]. [score:3]
Normalisation was carried out either to hsa-miR-425* (dark blue), RNU43 (red), hsa-miR-877 (green), RNU24 (purple) or geometric mean of hsa-miR-425* and RNU24 (light blue). [score:1]
Candidates again displayed low variability, with miR-877 exhibiting the smallest Cq variation (CV = 3.33), and RNU44 (CV = 6.60) the largest. [score:1]
The fold changes of all three miRNAs normalised against RNU43 was different from the fold changes obtained following the normalisation against hsa-miR-425*, hsa-miR-877, RNU24 and the geometric mean of hsa-miR-425* and RNU24. [score:1]
For this analysis, normalisation of these three miRNAs was performed using top ranked candidate EC reference genes identified by geNorm and Normfinder, including hsa-miR-425*, RNU24, hsa-miR-877, and the EC reference pair, RNU24/ hsa-miR-425*. [score:1]
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7
[+] score: 9
The results are consistent with findings by Vickers and colleagues, indicating that miR-877 is abundantly expressed in normal individuals while miR-191 is up-regulated in familial hypercholesterolemia (FH) human subjects. [score:6]
miR-877 and 191 were differentially expressed in low and high LDL-C livers, respectively. [score:3]
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8
[+] score: 9
Eight up-regulated miRNAs, including miR-19a-3p, miR-877-3p, miR-148a-3p, miR-212-5p, miR-1825, miR-210-3p, miR-940, and miR-134-5p, and two down-regulated miRNAs, miR-3609 and miR-145-5p, were identified as statistically significant different miRNAs. [score:7]
No studies on the relationship between the other miRNAs, miR-877-3p, miR-212-5p, miR-1825, miR-940, miR-134-5p, and miR-3609, and PE have been reported. [score:1]
Considering that the conversion of the biological functions of normal cells is fundamental to the pathology of PE and carcinoma, we infer that miR-877-3p, miR-212-5p, miR-1825, miR-940, miR-134-5p, and miR-3609 might take part in the progression of PE. [score:1]
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9
[+] score: 7
Among the miRNAs, the function of fifteen downregulated miRNAs (miR-1587, miR-4505, miR-1915-3p, miR-4466, miR-3162-5p, miR-4484, miR-5001-5p, miR-3940-5p, miR-4687-3p, miR-6068, miR-548ao-3p, miR-4687-5p, miR-1273g-3p, miR-877-3p, miR-4312) and the function of two miRNAs that were upregulated (miR-520f-3p, miR-4307) remain unknown. [score:7]
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10
[+] score: 7
They identified 13 miRNAs that were differentially expressed in OSCC when compared to healthy controls and, among them, 11 miRNAs were down-regulated (miRNA-136, miRNA-147, miRNA-1250, miRNA-148a, miRNA-632, miRNA-646, miRNA-668, miRNA-877, miRNA-503, miRNA-220a, miRNA-323-5p), and two miRNAs were over-expressed (miRNA-24, miRNA-27b). [score:7]
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11
[+] score: 7
They have identified miR-483, miR-877, miR-337-5p, miR-546, and miR-494 as being upregulated, and miR-770-5p, miR-487b, miR-220, miR-212, and miR-712 as downregulated by adenosine signaling in MΦs. [score:7]
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12
[+] score: 6
Other miRNAs from this paper: hsa-mir-224, hsa-mir-141, hsa-mir-200c, hsa-mir-452, hsa-mir-888
The one microRNA that is significantly differentially expressed between the 6p+12p tensor GSVD classes, and maps to the same deletion, is the splicing -dependent microRNA miR-877*, which is encoded by the 13th intron of the ATP -binding cassette subfamily F member 1-encoding gene ABCF1 [44]. [score:3]
Both miR-877* and ABCF1 are consistently underexpressed. [score:3]
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13
[+] score: 6
Overexpression of hsa-miR-877-5p was observed in metastatic melanoma [23] and endometrial serous adenocarcinomas [24], but no biological function was determined. [score:3]
Despite the fact that the first splicing -dependent human mirtrons were annotated in 2007 [15] and a large number of splicing-derived miRNAs have been predicted by bioinformatic analysis of deep sequencing data [12, 14], only two of them, hsa-miR-877-5p and hsa-miR-1226-3p, were experimentally proven to be directly affected by splicing [16, 17]. [score:2]
Nevertheless, their dependence on splicing was experimentally validated only for two human miRNAs located in conventional mirtrons, hsa-miR-877-5p and hsa-miR-1226-3p [16, 17]. [score:1]
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14
[+] score: 5
MDM4 | miR-191 or miR-877-3pMdm2-like p53 -binding protein (MDM4) is an oncoprotein that negatively regulates the p53 tumour suppressor protein [70]. [score:4]
MDM4 | miR-191 or miR-877-3p. [score:1]
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15
[+] score: 5
Still, statistical analysis detected only 11 miRNAs with altered expression in HeLa cells, of which only four miRNAs (hsa-miR-146b, hsa-miR-577, hsa-miR-877 and hsa-miR-193b*) had FC > 2 expression change between cell cycle phases. [score:5]
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16
[+] score: 5
In addition, besides expression of 18 EBV-BART transcripts, 10 cellular miRNAs (miR-16, miR-26a, miR-142-5p, miR-148a, miR-200b, miR-223, miR-668, miR-877, miR-1178, and miR-1233) were shown to have an elevated expression pattern in EBV positive ID-BL [75]. [score:5]
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17
[+] score: 4
Among all of the TGF-β pathway-related miRNAs, five known miRNAs (miR-3180-3p, miR-34b-5p, miR-877-3p, miR-936, and miR-940) and 10 novel miRNA candidates (seq-915_x4024, seq-5118_x304, seq-6713_x208, seq-14465_x69, seq-18595_x48, seq-19788_x44, seq-38785_x17, seq-38875_x17, seq-48658_x13, and seq-52107_x11) target pathway members that positively regulate the pathway. [score:4]
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18
[+] score: 3
miR-871 - ND miR-875-3p - - - ND miR-877 + ND miR-883a-5p - ND miR-93 +miR-93 was over-expressed in human T-cell leukemia virus 1- transformed human T-cell lines and primary peripheral blood mononuclear cells from adult T-cell leukemia patients [46]. [score:3]
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19
[+] score: 3
10 miRNAs were consistently validated (Fig.   6) to be differentially expressed by HP diet, namely miR-592, miR-488-3p, miR-339-3p, miR-17-5p-1, miR-877-5p, miR-15b-5p, miR-484-1, miR-4745-5p, let-7b-5p and miR-25-3p. [score:3]
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20
[+] score: 3
34 hsa-miR-335 −0.35hsa-miR-345 [44], [53], [71] 1.16 hsa-miR-363 0.99 hsa-miR-371-5p 0.55 hsa-miR-421 0.50 hsa-miR-483-5p 1.33 hsa-miR-494 0.87 hsa-miR-505* −0.40 hsa-miR-513a-5p 1.06 hsa-miR-513b 1.19 hsa-miR-513c 1.22 hsa-miR-551b −0.40 hsa-miR-574-5p 0.97hsa-miR-630 [68], [73] 0.96 hsa-miR-769-5p −0.34 hsa-miR-801 0.66 hsa-miR-873 −0.64 hsa-miR-877* 0.72 hsa-miR-923 0.89 hsa-miR-940 0.49 hsa-miR-95 −0.44 hsa-miR-99a −0.64Irradiated and non-irradiated PBL of the same donors were incubated in static gravity (1 g) for 4 and 24 h, and miRNA expression profile was analyzed at the end of each incubation time. [score:3]
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21
[+] score: 3
miRNA Fold change at 3 dpi Fold change at 5 dpi mmu-miR-466h-3p NS (Not significant) 14.311053 mmu-miR-346-5p NS 3.4766614 mmu-miR-877-3p NS 3.416667 mmu-miR-7a-5p NS 2.1413074 mmu-miR-5107-5p NS −2.047792 mmu-miR-3473a −2.2872427 −2.1317267 mmu-miR-150-5p NS −2.1770155 mmu-miR-3473b −3.2475147 −2.282881 mmu-miR-721 NS −2.6864858 mmu-miR-669b-5p NS −2.9408455 mmu-miR-709 NS −3.0065749 mmu-miR-669n NS −3.0094464 mmu-miR-468-3p NS −3.40051 mmu-miR-466m-5p NS −4.33538 mmu-miR-32-3p NS −4.5324426 mmu-miR-466h-5p NS −4.9673104 mmu-miR-3082-5p NS −6.01648 mmu-miR-466i-5p NS −7.6776285 mmu-miR-1187 NS −8.772696 mmu-miR-574-5p NS −9.259378 To confirm the validity of the differentially expressed miRNAs that had been identified by microarray analysis, we performed real-time PCR on all 20 of these miRNAs using the polyA tailing technique. [score:3]
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22
[+] score: 3
In addition, only miR-877* were common to both studies and had consistent direction of regulation following 2009 pandemic H1N1 influenza virus infection in two studies. [score:3]
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23
[+] score: 3
Other miRNAs specific to MAB-S-infected cells have also been found to differ in their expression levels between different strains of MTB (miR-99b and miR-140) or between latent and active tuberculosis (miR-877) [54]. [score:3]
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24
[+] score: 1
The chromosomes HSCHR6_MHC_COX, HSCHR6_MHC_MANN, HSCHR6_MHC_DBB, HSCHR6_MHC_MCF, HSCHR6_MHC_QBL containing hsa-mir-219-1, HSCHR6_MHC_COX, HSCHR6_MHC_DBB, HSCHR6_MHC_MCF, HSCHR6_MHC_MANN, HSCHR6_MHC_QBL, HSCHR6_MHC_SSTO containing hsa-mir-877, HSCHR6_MHC_COX, HSCHR6_MHC_DBB, HSCHR6_MHC_MCF, HSCHR6_MHC_QBL, HSCHR6_MHC_SSTO containing hsa-mir-1236, and MT containing hsa-mir-1974, hsa-mir-1977 and hsa-mir-1978 as mentioned in the miRBase have been discarded. [score:1]
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25
[+] score: 1
90 Down 3.84E-18 hsa-miR-4448 2.45 Up 1.66E-04 hsa-miR-1250 −5.71 Down 2.05E-169 hsa-miR-4426 2.23 Up 3.53E-05 hsa-miR-139-5p −5.46 Down 3.71E-25 hsa-miR-720 2.08 Up 1.21E-05 hsa-miR-335-5p −5.26 Down 2.00E-41 hsa-miR-4485 1.71 Up 6.14E-08 hsa-miR-362-3p −5.01 Down 3.36E-10 hsa-miR-877-5p 1.68 Up 1.92E-82 hsa-miR-1255a −4.92 Down 1.25E-62 hsa-miR-3679-5p 1.45 Up 5.53E-04 hsa-miR-3622a-5p −4.83 Down 4.30E-09 hsa-miR-21-3p 1.40 Up 7.87E-13 hsa-miR-1293 −4.67 Down 2.87E-08 hsa-miR-2116-3p −4.64 Down 3.67E-14 hsa-miR-365a-3p −4. [score:1]
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26
[+] score: 1
We found: miR-4290, miR-1281, miR-4716-5p, miR-483-3p and miR-877-3p containing almost only UC nucleotides and miR-6124, miR-483-5p, miR-4271, miR-4644, miR-4716-3p and miR-1234-5p purine-rich. [score:1]
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27
[+] score: 1
Other miRNAs from this paper: hsa-mir-1249, hsa-mir-3167, hsa-mir-5001
shtml) as described in the literature [34], we found that lncRNA RPAIN contains potential binding sites for the following 4 miRNAs: miR-3167, miR-1249-5p, miR-877-5p, and miR-5001-5p. [score:1]
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28
[+] score: 1
The respective 16 markers include hsa-miR-1299, hsa-miR-3124, hsa-miR-4290, hsa-miR-2278, hsa-miR-32*, hsa-miR-3149, hsa-miR-877*, hsa-miR-584, hsa-miR-3148, hsa-miR-122, hsa-miR-718, hsa-miR-670, hsa-miR-3680*, hsa-miR-193b*, hsa-miR-3911 and hsa-miR-1228* (see Figure  4). [score:1]
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29
[+] score: 1
The present study uncovered only one SNP (rs35730843, POLR2G, P = 1.2 × 10 [-5], OR = 0.393) strongly associated with PBC and 11 SNPs strongly associated with PSC (rs3822659, coding in WWC1, P = 0.0051, OR = 0.236; rs9686714, intron of WWC1, P = 0.00077, OR = 0.195; rs13191240, intron of ADGRB3, P = 0.0095, OR = 0.2; rs7454108, intergenic between LOC100294145 and C4B_2, P = 0.0013, OR = 0.326; rs2524163, intron of HLA-B, P = 5.9 × 10 [-7], OD = 2.02; rs2187668, intron of HLA-DQA1, P = 1.5 × 10 [-7], OR = 2.47; rs3130484, intron of MSH5-SAPCD1, P = 5.1 × 10 [-11], OR = 3.23; rs1264377, intergenic between PSORS1C3 − MIR877, P = 8 × 10 [-8], OR = 2.39; rs3130626, intron of PRRC2A, P = 1.5 × 10 [-6], OR = 2.10; rs419788, intron of SKIV2L, P = 1.2 × 10 [-6], OR = 2.03; and rs34708188, intergenic close to SENCR, P = 0.0056, OR = 2.27). [score:1]
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30
[+] score: 1
Note that there is not evidence for small RNA generation across most of the aligned species, and only in a small number of the species exhibit a classic "saddle-shaped" evolutionary profile in which the hairpin loop clearly evolves more quickly than does the hairpin arms (e. g. as is seen for mir-1224, mir-3064, and mir-877). [score:1]
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31
[+] score: 1
Interestingly, three mirtrons (miR-877, miR-1224, miR-1225) were included. [score:1]
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