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miRBase |
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![]() 4 publications mentioning ocu-mir-34aOpen access articles that are associated with the species Oryctolagus cuniculus and mention the gene name mir-34a. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary. |
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Other miRNAs from this paper: hsa-mir-21, hsa-mir-92a-1, hsa-mir-92a-2, mmu-mir-146a, mmu-mir-155, hsa-mir-192, mmu-mir-199a-1, hsa-mir-199a-1, mmu-mir-122, hsa-mir-34a, hsa-mir-199a-2, hsa-mir-223, hsa-mir-122, hsa-mir-146a, mmu-mir-192, mmu-mir-21a, mmu-mir-92a-2, mmu-mir-34a, hsa-mir-155, mmu-mir-223, mmu-mir-199a-2, mmu-mir-92a-1, hsa-mir-146b, hsa-mir-92b, mmu-mir-146b, mmu-mir-92b, mmu-mir-21b, mmu-mir-21c, ocu-mir-21, ocu-mir-92a-1, ocu-mir-92a-2, ocu-mir-122, ocu-mir-146a, ocu-mir-146b, ocu-mir-155, ocu-mir-199a-1, ocu-mir-199a-2, ocu-mir-223
For example, miR-146b and miR-34a were up-regulated in the liver tissues of patients with non-alcoholic steatohepatitis [11], while in the CCl [4] induced liver fibrosis, miR-199a-5p and miR-199a-3p were positively and significantly correlated to the progression of liver fibrosis [12].
[score:4]
To identify miRNAs that reflected the schistosome infections and PZQ chemotherapy, six miRNA candidates (miR-146b, miR-122, miR-223, miR-199a-5p, miR-199a-3p, miR-34a) were selected for analysis in serum that were commonly deregulated in human liver diseases.
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Expression levels of serum miR-223 (B), miR-122 (C), miR-34a (D), miR-199a-5p (E) miR-199a-3p (F), and miR-146b (G) were detected in the three groups of mice.
[score:3]
The expression levels of miR-34a, miR-223, miR-122, miR-146b, miR-199a-5p, miR-199a-3p were determined using the SYBR Green Master Mix kit (TaKaRa, Dalian, China).
[score:3]
In mouse hosts, quantitative PCR result revealed that circulating miR-223, miR-122 and miR-34a were significantly elevated after infection (FigureĀ 1B-D).
[score:1]
We analyzed the serum levels of six selected candidate miRNA molecules (miR-146b, miR-122, miR-223, miR-199a-5p, miR-199a-3p, miR-34a) from mice, rabbits, buffalos and humans infected with Schistosoma japonicum using qPCR.
[score:1]
To test this hypothesis, we selected six candidate serum miRNAs for analysis (miR-146b, miR-122, miR-223, miR-199a-5p, miR-199a-3p, miR-34a) in the murine mo del of human schistosomiasis and then performed validation in other host species including rabbits, buffalos and human patients infected with S. japonicum.
[score:1]
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Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-21, hsa-mir-23a, hsa-mir-26a-1, hsa-mir-26b, hsa-mir-92a-1, hsa-mir-92a-2, hsa-mir-98, hsa-mir-199a-1, hsa-mir-7-1, hsa-mir-7-2, hsa-mir-7-3, hsa-mir-10b, hsa-mir-34a, hsa-mir-199a-2, hsa-mir-203a, hsa-mir-214, hsa-mir-221, hsa-mir-223, hsa-mir-224, hsa-mir-200b, hsa-let-7g, hsa-let-7i, hsa-mir-23b, hsa-mir-122, hsa-mir-125b-1, hsa-mir-141, hsa-mir-191, hsa-mir-125b-2, hsa-mir-126, hsa-mir-127, hsa-mir-200c, hsa-mir-26a-2, hsa-mir-638, hsa-mir-762, hsa-mir-1275, hsa-mir-203b, ocu-mir-191, ocu-let-7a-1, ocu-let-7a-2, ocu-let-7a-3, ocu-let-7b, ocu-let-7c, ocu-let-7d, ocu-let-7f-1, ocu-let-7f-2, ocu-let-7g, ocu-let-7i, ocu-mir-7a, ocu-mir-10b, ocu-mir-21, ocu-mir-23b, ocu-mir-26a, ocu-mir-26b, ocu-mir-92a-1, ocu-mir-92a-2, ocu-mir-98, ocu-mir-122, ocu-mir-125b-1, ocu-mir-125b-2, ocu-mir-127, ocu-mir-141, ocu-mir-199a-1, ocu-mir-199a-2, ocu-mir-200c, ocu-mir-214, ocu-mir-221, ocu-mir-223, ocu-mir-224
In this study, we found that, in addition to lowered levels of miR-122, expression of miR-223, miR-34a, and miR-127 was also down-regulated.
[score:6]
miR-223 is noteworthy because it is frequently suppressed in human HCC [52], and miR-34a, miR-127 and miR-200b are down-regulated in the rat liver during experimental hepatocarcinogenesis [36].
[score:6]
Tryndyak V. P. Ross S. A. Beland F. A. Pogribny I. P. Down-regulation of the microRNAs miR-34a, miR-127, and miR-200b in rat liver during hepatocarcinogenesis induced by a methyl -deficient diet Mol.
[score:4]
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Other miRNAs from this paper: mmu-mir-9-2, mmu-mir-140, mmu-mir-146a, mmu-mir-10b, hsa-mir-196a-1, hsa-mir-10b, hsa-mir-34a, hsa-mir-196a-2, hsa-mir-221, hsa-mir-140, hsa-mir-9-1, hsa-mir-9-2, hsa-mir-9-3, hsa-mir-146a, mmu-mir-196a-1, mmu-mir-196a-2, mmu-mir-34a, mmu-mir-221, mmu-mir-9-1, mmu-mir-9-3, hsa-mir-196b, mmu-mir-196b, gga-mir-221, gga-mir-196-2, gga-mir-10b, gga-mir-140, gga-mir-146a, gga-mir-34a, gga-mir-196-1, gga-mir-196-3, gga-mir-9-2, gga-mir-9-1, gga-mir-196-4, gga-mir-196-5, gga-mir-9-3, ocu-mir-9-1, ocu-mir-9-2, ocu-mir-10b, ocu-mir-140, ocu-mir-146a, ocu-mir-196a-1, ocu-mir-196a-2, ocu-mir-196b, ocu-mir-221, gga-mir-9-4, mmu-mir-9b-2, mmu-mir-9b-1, mmu-mir-9b-3, gga-mir-9b-1, gga-mir-9b-2
We reported the involvement of several miRNAs including miR-34a [9, 10] and miR-221 [11] in JNK-regulated chondrogenic differentiation.
[score:2]
Our laboratory also showed that JNK signaling is involved in the differentiation of chondroprogenitors in chicks through regulation of miR-34a [9, 10] and miR-221 levels [11].
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[34] Recent reports also showed that several miRNAs (miR-34a) and transcription factors (β-catenin and slug) regulate CD171 expression in endometrial adenocarcinomas.
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