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35 publications mentioning hsa-mir-105-1

Open access articles that are associated with the species Homo sapiens and mention the gene name mir-105-1. Click the [+] symbols to view sentences that include the gene name, or the word cloud on the right for a summary.

1
[+] score: 14
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-17, hsa-mir-19a, hsa-mir-21, hsa-mir-31, hsa-mir-92a-1, hsa-mir-92a-2, hsa-mir-105-2, hsa-mir-199a-1, hsa-mir-34a, hsa-mir-187, hsa-mir-199a-2, hsa-mir-205, hsa-mir-214, hsa-mir-221, hsa-let-7g, hsa-let-7i, hsa-mir-128-1, hsa-mir-141, hsa-mir-144, hsa-mir-145, hsa-mir-9-1, hsa-mir-9-2, hsa-mir-9-3, hsa-mir-146a, hsa-mir-200c, hsa-mir-128-2, hsa-mir-29c, hsa-mir-376a-1, hsa-mir-378a, hsa-mir-133b, hsa-mir-429, hsa-mir-487a, hsa-mir-515-1, hsa-mir-515-2, hsa-mir-526b, hsa-mir-514a-1, hsa-mir-514a-2, hsa-mir-514a-3, hsa-mir-376a-2, hsa-mir-548a-1, hsa-mir-548b, hsa-mir-548a-2, hsa-mir-548a-3, hsa-mir-548c, hsa-mir-548d-1, hsa-mir-548d-2, hsa-mir-656, hsa-mir-542, hsa-mir-378d-2, hsa-mir-548e, hsa-mir-548j, hsa-mir-548k, hsa-mir-548l, hsa-mir-548f-1, hsa-mir-548f-2, hsa-mir-548f-3, hsa-mir-548f-4, hsa-mir-548f-5, hsa-mir-548g, hsa-mir-548n, hsa-mir-548m, hsa-mir-548o, hsa-mir-548h-1, hsa-mir-548h-2, hsa-mir-548h-3, hsa-mir-548h-4, hsa-mir-1275, hsa-mir-548p, hsa-mir-548i-1, hsa-mir-548i-2, hsa-mir-548i-3, hsa-mir-548i-4, hsa-mir-2114, hsa-mir-548q, hsa-mir-548s, hsa-mir-378b, hsa-mir-548t, hsa-mir-548u, hsa-mir-548v, hsa-mir-548w, hsa-mir-548x, hsa-mir-514b, hsa-mir-378c, hsa-mir-4303, hsa-mir-4309, hsa-mir-4307, hsa-mir-4278, hsa-mir-548y, hsa-mir-548z, hsa-mir-548aa-1, hsa-mir-548aa-2, hsa-mir-548o-2, hsa-mir-378d-1, hsa-mir-378e, hsa-mir-548h-5, hsa-mir-548ab, hsa-mir-378f, hsa-mir-378g, hsa-mir-548ac, hsa-mir-548ad, hsa-mir-548ae-1, hsa-mir-548ae-2, hsa-mir-548ag-1, hsa-mir-548ag-2, hsa-mir-548ah, hsa-mir-378h, hsa-mir-548ai, hsa-mir-548aj-1, hsa-mir-548aj-2, hsa-mir-548x-2, hsa-mir-548ak, hsa-mir-548al, hsa-mir-378i, hsa-mir-548am, hsa-mir-548an, hsa-mir-548ao, hsa-mir-548ap, hsa-mir-548aq, hsa-mir-548ar, hsa-mir-548as, hsa-mir-548at, hsa-mir-548au, hsa-mir-548av, hsa-mir-548aw, hsa-mir-548ax, hsa-mir-378j, hsa-mir-548ay, hsa-mir-548az, hsa-mir-548ba, hsa-mir-548bb, hsa-mir-548bc
The aberrantly expression miRNAs such as has-miR-214*, has-miR-105, has-miR-548n, and has-miR-514 which are upregulate in gastric cancerous tissues. [score:6]
The expression level of miR-214* is increased 3.79-fold (Figure 4(a)), miR-105 is increased 16.32-fold (Figure 4(b)), has-miR-548 is 4.21-fold (Figure 4(c)), and miR-514 is increased 11.76-fold (Figure 4(d)) in comparing with normal tissues. [score:3]
The has-miR-105 was highly expressed in testis tumors [32]. [score:3]
We selected hsa-miR-105, hsa-miR-214*, hsa-miR-514b, and has-miR-548n to test in 24 paired gastric normal and tumor tissues. [score:1]
Comparing with previous studies of miRNAs, we found that 21 among 31 genes have been reported in previous publications, for example, hsa-miR-105, hsa-miR-187, hsa-miR-214*, hsa-miR-656, and hsa-miR-487a. [score:1]
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2
[+] score: 13
In the formalin-fixed squamous cell carcinomas of the tongue, hsa-miR-105 is one of the up-regulated miRNAs [24], and highly expressed in seminoma [25]. [score:6]
The other four miRNAs, hsa-miR-942, hsa-miR-105, hsa-miR-150, and hsa-miR-27a* were identified to be up-regulated in ovarian cancer. [score:4]
In all, 5 nodes with degrees more than 30, including hsa-miR-579 (36), hsa-miR-942 (31), hsa-miR-105 (31), hsa-miR-150 (34), and hsa-miR-27a* (32) were selected as the hub nodes in MFSN. [score:1]
According to their degrees in MFSN, hsa-miR-579, hsa-miR-942, hsa-miR-105, hsa-miR-150, and hsa-miR-27a* were selected as hub nodes in MFSN. [score:1]
GO: 0007268 (synaptic transmission) and GO: 0019226 (transmission of nerve impulse) were the two common functions of miRNAs in MFSN, and hsa-miR-579 (36), hsa-miR-942 (31), hsa-miR-105 (31), hsa-miR-150 (34), and hsa-miR-27a* (32) were selected as the hub nodes in MFSN. [score:1]
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3
[+] score: 13
For example, miR-105 has been reported to function as a potential tumor suppressor and inhibit cell proliferation by regulating the PI3K/AKT signaling pathway in human HCC [11]; Tan and colleagues identified a serum of miRNA panel (hsa-miR-206, hsa-miR-141-3p, hsa-miR-433-3p, hsa-miR-1228-5p, hsa-miR-199a-5p, hsa-miR-122-5p, hsa-miR-192-5p, and hsa-miR-26a-5p) that has considerable clinical value in HCC diagnosis [12]; The presence of miR-101 has also been indicated to be a biochemical marker for monitoring the progression of tumor development in HBV-related HCC, and to be a potential prognostic marker and therapeutic target for HCC [13]. [score:9]
Shen G Rong X Zhao J Yang X Li H Jiang H Zhou Q Ji T Huang S Zhang J Jia H MicroRNA-105 suppresses cell proliferation and inhibits PI3K/AKT signaling in human hepatocellular carcinomaCarcinogenesis 2014 12. [score:4]
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4
[+] score: 9
Overexpression of miR-105 in non-metastatic cancer cells induces metastasis and vascular permeability in distant organs, whereas inhibition of miR-105 in highly metastatic tumors alleviates these effects [91]. [score:5]
miR-105, which is characteristically expressed and secreted by metastatic breast cancer cells, is a potent regulator of migration through targeting the tight junction protein ZO-1. In endothelial monolayers, exosome -mediated transfer of cancer-secreted miR-105 efficiently destroys tight junctions and the integrity of these natural barriers against metastasis. [score:4]
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5
[+] score: 9
Other miRNAs from this paper: hsa-mir-105-2, hsa-mir-223, hsa-mir-760
To verify whether ARTN expression is regulated by miRNAs, miR-105, miR-223 and miR-760 were chosen for further study according to the results of a bioinformatic search. [score:4]
miR-105, miR-223 and miR-760 including a 200 bp flanking sequence were cloned from human genomic DNA and inserted into pcDNA3.1 (+) multiple cloning sites to construct the miRNAs expression vectors. [score:3]
As a result, the three miRNAs with the highest free energy (hsa-mir-105, hsa-mir-223 and hsa-mir-760) were chosen. [score:1]
The genomic sequences, including 200 bp flanking sequences, of the human miR-105, miR-223 and miR-760 genes were cloned from HEK293 cells. [score:1]
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6
[+] score: 8
A set of ADAMTS enzymes (ADAMTS4, ADAMTS5, ADAMTS7, as well as ADAMTS12) are controlled by miRNA-105, plausibly via targeting Runt-related transcription factor 2 (Runx2). [score:3]
Ji Q. Xu X. Xu Y. Fan Z. Kang L. Li L. Liang Y. Guo J. Hong T. Li Z. miR-105/Runx2 axis mediates FGF2 -induced ADAMTs expression in osteoarthritis cartilage J. Mol. [score:3]
Significantly, it has been reported that there is an inverse correlation among miRNA-105 and Runx2, as well as ADAMTS [89]. [score:1]
In turn, miRNA-105 is negatively modulated by the p65 subunit of NF-kB upon stimulation of chondrocytes with fibroblast growth factor 2 (FGF2), suggesting that an FGF2/p65/miRNA-105/Runx2/ADAMTS pathway is critically implicated in the etiopathogenesis of OA. [score:1]
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7
[+] score: 7
In the same way, miR-105 suppresses cell proliferation by downregulating insulin receptor substrate-1, 3-phosphoinositide -dependent protein kinase-1, and AKT1 directly [41]. [score:7]
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8
[+] score: 7
42 members of the miR-515 family (miR-515–527) and eight members of the miR-302–367 cluster (miR-302a, miR-302a*, miR-302b, miR-302b* miR-302c, miR-302d, miR-302d* and miR-367) were only up-regulated in TGCT and not in the other two cancers, and three members of the miR-105-767 cluster (miR-105, miR-105* and miR-767-5p) were ranked as the top ten up-regulated miRNAs in TGCT. [score:7]
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9
[+] score: 7
Other miRNAs from this paper: hsa-mir-105-2, hsa-mir-203a, hsa-mir-155, hsa-mir-203b
Gingival human oral keratinocytes incubated with heat-inactivated P. gingivalis exhibited up-regulation of miRNA-105, which is complementary to TLR2 mRNA (Benakanakere et al., 2009). [score:4]
Modulation of TLR2 protein expression by miR-105 in human oral keratinocytes. [score:3]
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10
[+] score: 6
miR-105, for example, has been shown to regulate the protein expression of TLR2 in human keratinocytes [52], while miR-182 expression is a biomarker for patients with sepsis [53]. [score:6]
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11
[+] score: 6
For instance, miR-146 downregulates TRAF6, IRAK1 and IRAK2 while, let-7i and miR-105 target TLR4 and TLR2, respectively [12– 15]. [score:6]
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12
[+] score: 5
Of these, we highlighted 10 miRNAs (miR-9, miR-145*, miR-105, miR-147b, let-7f-2*, let-7i*, miR-302c*, miR-199a-3p, miR-222* and miR-942) whose expression was significantly higher at P<0.0005 (Fig. 3A ). [score:3]
The sensitivity level for miR-145*, miR-105, and miR-942 was 77.8%, and that for miR-147b, let-7f-2*, and let-7i* was 66.7% (Fig. 3B ). [score:1]
Setting the specificity threshold to 100% showed the sensitivity level to be 88.9% for miR-9, miR-302c*, miR-199a-3p, and miR-222*; 77.8% in miR-145*, miR-105, and miR-942; and 66.7% in miR-147b, let-7f-2*, and let-7i*. [score:1]
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13
[+] score: 5
However, although TLR2 is targeted by several human miRNAs including miR-105, miR-19, miR-1225-5p, miR-143 and miR-154 in a variety of cell types [38– 42], no viral miRNA has been shown to target TLR2—until now. [score:5]
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14
[+] score: 4
Our miRNA profile (84) Chromosomal localization Fold change Reference DOWNREGULATED hsa-miR-206 6p12.2 −7.53(29) hsa-miR-219-2-3p 9q33.3 −6.64(52) hsa-miR-383 8p22 −6.56(12, 55, 56) hsa-miR-138 16q13.3/3p21.32 −5.16(12, 14) hsa-miR-323-3p 14q32.2 −4.96(12, 52) hsa-miR-122 18q21.31 −4.82 hsa-miR-105 Xq28 −4.66 hsa-miR-129-5p 11p11.2/7q32.1 −4.56(23) hsa-miR-935 19q13.43 −4.53(52) hsa-miR-329 14q32.2 −4.48 hsa-miR-129-3p 11p11.2/7q32.1 −4.43 hsa-miR-650 22q11.21 −4.19 hsa-miR-184 15q24.3 −4.14 hsa-miR-370 14q32.2 −3.99(12) hsa-miR-433 14q32.2 −3.96(29) hsa-miR-138-2* 16q13. [score:4]
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15
[+] score: 4
Eight miRNAs such as miR-105 were upregulated in the diffuse type while only four miRNAs such as miR-373 increased in the intestinal type [92]. [score:4]
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16
[+] score: 4
Other miRNAs, such as miR-511, miR-21, miRNA-223, miRNA-105 and let-7e were also reported to be involved in the regulation of TLRs signal [13], [30], [31]. [score:2]
TLR2 protein has been shown to be regulated by miR-105 in human oral keratinocytes [14]. [score:2]
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17
[+] score: 4
Furthermore, the regulation of its expression through miR-19 and miR-105 has been described by other authors [44, 45]. [score:4]
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18
[+] score: 3
Novotny et al. performed a specific statistical analysis to understand how CIS cells progress and degenerate in malignant tumor by determining whether any changes in gene expression are statistically significant and they discovered that miR-9 and miR-105 levels were high in SE, miR-182, miR-183 and miR-96 levels were increased in SE and embryonal carcinoma, while miR-515-526 cluster was really increased both in CIS, SE and embryonal carcinoma [89] (see Table 3). [score:3]
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19
[+] score: 3
For instance, adipogenic differentiation was highly increased by miR105 and miR371 which was accompanied by respective gene expression changes of certain adipogenic key molecules including adiponectin and fatty acid -binding protein 4. Conversely, adipogenic differentiation of MSC can be impaired by miR-369-5p [53]. [score:3]
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20
[+] score: 3
Other miRNAs from this paper: hsa-mir-105-2
Further, curcumin treatment of EC produces exosomes that promote EC barrier function [57] while cancer-secreted exosomes containing miR-105 inhibit EC barrier function and promote metastasis [58]. [score:3]
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21
[+] score: 3
In particular, Bloomston and colleagues [34], using the miRNA microarray chip OSU_CCC v. 3.0, which contains 326 human miRNA probes, identified a subgroup of six miRNAs (miR-30a-3p, miR-105, miR-127, miR-187, miR-452, and miR-518a2) that distinguish long-term survivors with node -positive disease from those succumbing within 24 months. [score:3]
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22
[+] score: 3
And we selected SNPs located in various miRNAs, including miRNAs which regulate breast cancer -associated genes, such as RAS, PTEN, ATM and BRCA1/2 (hsa-mir-149, hsa-mir-196a-2, hsa-mir-30c-1, hsa-mir-146a, hsa-let-7f-2); miRNAs regulating breast cancer associated receptors ER, PR and HER2 (hsa-mir-27a, hsa-mir-125b-1, hsa-mir-105-1, hsa-mir-105-2); and miRNAs closely associated with breast caner invasion and metastasis (hsa-mir-373 and hsa-mir-10b). [score:3]
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23
[+] score: 3
More recently, Zhou et al showed that the transfer of exosomal miR-105 to non-metastatic breast cancer cells induces metastasis and vascular permeability by targeting the cellular tight junction protein ZO-1 (113). [score:3]
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24
[+] score: 3
In contrast, miRNAs, which are frequently detected in different cancers exosomes (include hsa-miR-105, hsa-miR-214, hsa-miR-92, hsa-miR-21, hsa-miR-29, hsa-miR-9, hsa-miR-222) 63 64, were either absent or showed very nominal expression in hAMSC-CM derived exosomes. [score:3]
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25
[+] score: 3
Bioinformatics analyses and cellular mo deling from the same group identified two additional microRNAs, miR-155 and miR-105, that target the 3′UTR of NPPA. [score:3]
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26
[+] score: 3
The targets of some miRNAs were strongly enriched in certain categories, e. g., miR-105 in “small GTPase mediated signal transduction” (5-fold), miR-208 in “transcription factor” (6-fold), and miR-7, which lies in the intron of the hnRNPk (an RNA -binding protein) gene, in “RNA binding proteins. [score:3]
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27
[+] score: 2
Other miRNAs from this paper: hsa-mir-105-2, mmu-mir-105, hsa-mir-1200, rno-mir-105
Search of this HAfT exon1 sequence shows alignment to rno-miR-105, inferring possible miR regulation. [score:2]
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28
[+] score: 1
In the remaining 46 miRNAs with a corresponding Affymetrix U133A probe for their pri-miRNA transcripts, the ratio of miRNA to host mRNA was significantly lower for two pri-miRNAs (primary transcripts for miR-218, p = 0.021, and miR-9, p = 0.006) and significantly higher for five (miR-482, p = 0.015; miR-190, p = 0.018; miR-105, p = 0.02; miR-148b, p = 0.027; miR-218, p = 0.02). [score:1]
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29
[+] score: 1
Other miRNAs from this paper: hsa-mir-105-2, hsa-mir-126, hsa-mir-574, hsa-mir-660
Functional classification of these biomarkers includes growth and proliferation (Ki-67, survivin, NGAL), invasion and metastasis (p53, MMP-9, SK1, DcR3, COX2, EZH2, microRNAs miR-105, and miR126), epithelial–mesenchymal transition (EMT) (WNT5A/B, Pea3), immune response (PD-L1), therapy resistance (HER2Δ16, pSTS3, KLK10), survival (miR-574-3p, miR-660-5p, PIWIL3, PIWIL4), and many others (35). [score:1]
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30
[+] score: 1
In the single miRNA analysis three microRNA were found to be significant in both datasets (hsa-miR-105, hsa-miR-190, hsa-miR-433), however, in the single gene analysis we could not find even one gene that stratify prognosis robustly in both datasets. [score:1]
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31
[+] score: 1
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-17, hsa-mir-25, hsa-mir-92a-1, hsa-mir-92a-2, hsa-mir-105-2, dme-mir-1, dme-mir-10, mmu-let-7g, mmu-let-7i, mmu-mir-1a-1, mmu-mir-124-3, mmu-mir-134, mmu-mir-10b, hsa-mir-10a, hsa-mir-10b, dme-mir-92a, dme-mir-124, dme-mir-92b, mmu-let-7d, dme-let-7, hsa-let-7g, hsa-let-7i, hsa-mir-1-2, hsa-mir-124-1, hsa-mir-124-2, hsa-mir-124-3, hsa-mir-134, mmu-let-7a-1, mmu-let-7a-2, mmu-let-7b, mmu-let-7c-1, mmu-let-7c-2, mmu-let-7e, mmu-let-7f-1, mmu-let-7f-2, mmu-mir-92a-2, hsa-mir-1-1, mmu-mir-1a-2, mmu-mir-10a, mmu-mir-17, mmu-mir-25, mmu-mir-124-1, mmu-mir-124-2, mmu-mir-92a-1, hsa-mir-379, mmu-mir-379, mmu-mir-412, gga-let-7i, gga-let-7a-3, gga-let-7b, gga-let-7c, gga-mir-92-1, gga-mir-17, gga-mir-1a-2, gga-mir-124a, gga-mir-10b, gga-let-7g, gga-let-7d, gga-let-7f, gga-let-7a-1, gga-mir-1a-1, gga-mir-124b, gga-mir-1b, gga-let-7a-2, gga-let-7j, gga-let-7k, dre-mir-10a, dre-mir-10b-1, dre-mir-430b-1, hsa-mir-449a, mmu-mir-449a, dre-let-7a-1, dre-let-7a-2, dre-let-7a-3, dre-let-7a-4, dre-let-7a-5, dre-let-7a-6, dre-let-7b, dre-let-7c-1, dre-let-7c-2, dre-let-7d-1, dre-let-7d-2, dre-let-7e, dre-let-7f, dre-let-7g-1, dre-let-7g-2, dre-let-7h, dre-let-7i, dre-mir-1-2, dre-mir-1-1, dre-mir-10b-2, dre-mir-10c, dre-mir-10d, dre-mir-17a-1, dre-mir-17a-2, dre-mir-25, dre-mir-92a-1, dre-mir-92a-2, dre-mir-92b, dre-mir-124-1, dre-mir-124-2, dre-mir-124-3, dre-mir-124-4, dre-mir-124-5, dre-mir-124-6, dre-mir-430b-2, dre-mir-430b-3, dre-mir-430b-4, dre-mir-430b-6, dre-mir-430b-7, dre-mir-430b-8, dre-mir-430b-9, dre-mir-430b-10, dre-mir-430b-11, dre-mir-430b-12, dre-mir-430b-13, dre-mir-430b-14, dre-mir-430b-15, dre-mir-430b-16, dre-mir-430b-17, dre-mir-430b-18, dre-mir-430b-5, dre-mir-430b-19, dre-mir-430b-20, hsa-mir-412, hsa-mir-511, dre-let-7j, hsa-mir-92b, hsa-mir-449b, gga-mir-449a, hsa-mir-758, hsa-mir-767, hsa-mir-449c, hsa-mir-802, mmu-mir-758, mmu-mir-802, mmu-mir-449c, mmu-mir-105, mmu-mir-92b, mmu-mir-449b, mmu-mir-511, mmu-mir-1b, gga-mir-1c, gga-mir-449c, gga-mir-10a, gga-mir-449b, gga-mir-124a-2, mmu-mir-767, mmu-let-7j, mmu-let-7k, gga-mir-124c, gga-mir-92-2, gga-mir-449d, mmu-mir-124b, gga-mir-10c, gga-let-7l-1, gga-let-7l-2
The results showed that hsa-mir-758 can be assigned to the hsa-mir-379,380, 411 family; hsa-mir-767 can be assigned to the hsa-mir-105 family; and hsa-mir-802 can be assigned to the hsa-mir-511 family. [score:1]
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[+] score: 1
Other miRNAs from this paper: hsa-let-7a-1, hsa-let-7a-2, hsa-let-7a-3, hsa-let-7b, hsa-let-7c, hsa-let-7d, hsa-let-7e, hsa-let-7f-1, hsa-let-7f-2, hsa-mir-15a, hsa-mir-17, hsa-mir-20a, hsa-mir-21, hsa-mir-22, hsa-mir-23a, hsa-mir-24-1, hsa-mir-24-2, hsa-mir-25, hsa-mir-27a, hsa-mir-30a, hsa-mir-93, hsa-mir-96, hsa-mir-99a, hsa-mir-100, hsa-mir-29b-1, hsa-mir-29b-2, hsa-mir-105-2, hsa-mir-30c-2, hsa-mir-30d, hsa-mir-10a, hsa-mir-181a-2, hsa-mir-181b-1, hsa-mir-181c, hsa-mir-182, hsa-mir-205, hsa-mir-212, hsa-mir-181a-1, hsa-mir-222, hsa-mir-224, hsa-let-7g, hsa-let-7i, hsa-mir-23b, hsa-mir-27b, hsa-mir-30b, hsa-mir-122, hsa-mir-125b-1, hsa-mir-132, hsa-mir-141, hsa-mir-145, hsa-mir-191, hsa-mir-9-1, hsa-mir-9-2, hsa-mir-9-3, hsa-mir-125a, hsa-mir-125b-2, hsa-mir-146a, hsa-mir-150, hsa-mir-184, hsa-mir-188, hsa-mir-320a, hsa-mir-181b-2, hsa-mir-30c-1, hsa-mir-302a, hsa-mir-34c, hsa-mir-30e, hsa-mir-302b, hsa-mir-302c, hsa-mir-302d, hsa-mir-371a, hsa-mir-372, hsa-mir-376a-1, hsa-mir-378a, hsa-mir-383, hsa-mir-339, hsa-mir-133b, hsa-mir-345, hsa-mir-425, hsa-mir-483, hsa-mir-146b, hsa-mir-202, hsa-mir-193b, hsa-mir-181d, hsa-mir-498, hsa-mir-518f, hsa-mir-518b, hsa-mir-520c, hsa-mir-518c, hsa-mir-518e, hsa-mir-518a-1, hsa-mir-518d, hsa-mir-518a-2, hsa-mir-503, hsa-mir-513a-1, hsa-mir-513a-2, hsa-mir-376a-2, hsa-mir-548a-1, hsa-mir-548b, hsa-mir-548a-2, hsa-mir-548a-3, hsa-mir-548c, hsa-mir-645, hsa-mir-548d-1, hsa-mir-548d-2, hsa-mir-320b-1, hsa-mir-320c-1, hsa-mir-320b-2, hsa-mir-378d-2, hsa-mir-744, hsa-mir-548e, hsa-mir-548j, hsa-mir-548k, hsa-mir-548l, hsa-mir-548f-1, hsa-mir-548f-2, hsa-mir-548f-3, hsa-mir-548f-4, hsa-mir-548f-5, hsa-mir-548g, hsa-mir-548n, hsa-mir-548m, hsa-mir-548o, hsa-mir-548h-1, hsa-mir-548h-2, hsa-mir-548h-3, hsa-mir-548h-4, hsa-mir-302e, hsa-mir-302f, hsa-mir-548p, hsa-mir-548i-1, hsa-mir-548i-2, hsa-mir-548i-3, hsa-mir-548i-4, hsa-mir-320d-1, hsa-mir-320c-2, hsa-mir-320d-2, hsa-mir-548q, hsa-mir-548s, hsa-mir-378b, hsa-mir-548t, hsa-mir-548u, hsa-mir-548v, hsa-mir-548w, hsa-mir-320e, hsa-mir-548x, hsa-mir-378c, hsa-mir-548y, hsa-mir-548z, hsa-mir-548aa-1, hsa-mir-548aa-2, hsa-mir-548o-2, hsa-mir-378d-1, hsa-mir-378e, hsa-mir-548h-5, hsa-mir-548ab, hsa-mir-378f, hsa-mir-378g, hsa-mir-548ac, hsa-mir-548ad, hsa-mir-548ae-1, hsa-mir-548ae-2, hsa-mir-548ag-1, hsa-mir-548ag-2, hsa-mir-548ah, hsa-mir-378h, hsa-mir-548ai, hsa-mir-548aj-1, hsa-mir-548aj-2, hsa-mir-548x-2, hsa-mir-548ak, hsa-mir-548al, hsa-mir-378i, hsa-mir-548am, hsa-mir-548an, hsa-mir-371b, hsa-mir-548ao, hsa-mir-548ap, hsa-mir-548aq, hsa-mir-548ar, hsa-mir-548as, hsa-mir-548at, hsa-mir-548au, hsa-mir-548av, hsa-mir-548aw, hsa-mir-548ax, hsa-mir-378j, hsa-mir-548ay, hsa-mir-548az, hsa-mir-548ba, hsa-mir-548bb, hsa-mir-548bc
Validation of effects of selected pre-miR-10a, miR-105, and miR-182 by using other markers of proliferation (cyclin B1) and apoptosis (TdT and caspase 3) confirmed the specific role of miRNAs in human granulosa cell proliferation and apoptosis [47]. [score:1]
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For example, exosome -mediated transfer of cancer-secreted miR-105 has been shown to reduce endothelial barrier function; thereby promoting metastasis 43. [score:1]
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Zhou W Fong MY Min Y Somlo G Liu L Palomares MR Cancer-Secreted miR-105 destroys vascular endothelial barriers to promote metastasisCancer Cell. [score:1]
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Zhou W Cancer-secreted miR-105 destroys vascular endothelial barriers to promote metastasisCancer Cell. [score:1]
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